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Epilepsy Research|December 1, 1996
Chronic epilepsy with damage restricted to the hippocampus: possible mechanismsC G Wasterlain, Y Shirasaka, A M Mazarati, et al.Epilepsia|January 1, 1993
Pathophysiological mechanisms of brain damage from status epilepticusC G Wasterlain, D G Fujikawa, L Penix, et al.Brain Research|May 9, 1994
Nitric oxide participates in excitotoxic mechanisms induced by chemical hypoxiaT Akira, D Henry, R A Baldwin, et al.Brain Research|May 22, 1989
[3H]MK-801 binding sites in neonate rat brainA M Morin, H Hattori, C G Wasterlain, et al.Brain Research|August 21, 1989
Dark-induced changes in activity and compartmentalization of retinal calmodulin kinase in the ratJ Bronstein, C G Wasterlain, R Lasher, et al.Acta Neurologica Scandinavica. Supplementum|September 6, 2007
Advances in the pathophysiology of status epilepticusJ W Y Chen, D E Naylor, C G WasterlainBrain Research|September 5, 1998
Self-sustaining status epilepticus after brief electrical stimulation of the perforant pathA M Mazarati, C G Wasterlain, R Sankar, et al.European Journal of Pharmacology|March 3, 1992
Felbamate reduces hypoxic-ischemic brain damage in vivoC G Wasterlain, L M Adams, H Hattori, et al.Molecular Neurobiology|November 19, 2018
Early Aberrant Growth of Mossy Fibers after Status Epilepticus in the Immature Rat BrainA Rami, J Niquet, A KonoplewNeurochemical Research|December 7, 2010
Translocation of the serine protease Omi/HtrA2 from mitochondria into the cytosol upon seizure-induced hippocampal injury in the neonatal rat brainA Rami, M Kim, J NiquetPageof 20