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The American Journal of Physiology|February 1, 1989
Hypertension and alterations in central catecholamines after preoptic recess lesionsJ W Van Huysse, S L BealerThe American Journal of Physiology|June 1, 1992
Central nervous system catecholamine content and norepinephrine release after AV3V ablationJ W Van Huysse, S L BealerThe American Journal of Physiology|July 1, 1989
Axon destruction and adrenergic systems mediate pressor responses after AV3V lesionsS L Bealer, J W Van HuysseThe American Journal of Physiology|June 1, 1991
Central nervous system norepinephrine release during hypotension and hyperosmolality in conscious ratsJ W Van Huysse, S L BealerCellular & Molecular Biology Research|January 1, 1993
Nonpolar amino acid substitutions of potential cation binding residues glu-955 and glu-956 of the rat alpha 1 isoform of Na+, K(+)-ATPaseJ W Van Huysse, J B LingrelClinical and Experimental Hypertension (New York, N.Y. : 1993)|July 31, 1998
Role of endogenous brain "ouabain" in the sympathoexcitatory and pressor effects of sodiumJ W Van Huysse, F H LeenenJournal of Comparative and Physiological Psychology|April 1, 1978
Intensity coding in the transient portion of the rat chorda tympani responseS L BealerJournal of the Autonomic Nervous System|September 12, 1996
Preoptic recess lesions reduce right atrial pressure responses to volume expansionS L BealerBrain Research|January 16, 1997
Acute hypertension increases norepinephrine release in the diagonal band of BrocaS L BealerAmerican Journal of Physiology. Regulatory, Integrative and Comparative Physiology|May 9, 2000
Central control of cardiac baroreflex responses during peripheral hyperosmolalityS L BealerPageof 8