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Proceedings of the National Academy of Sciences of the United States of America|March 26, 2003
Macula densa cell signaling involves ATP release through a maxi anion channelPhillip Darwin Bell, Jean-Yves Lapointe, Ravshan Sabirov, et al.The New England Journal of Medicine|March 26, 2010
A loss-of-function mutation in NaPi-IIa and renal Fanconi's syndromeDaniella Magen, Liron Berger, Michael J Coady, et al.Plos One|June 1, 2013
Potassium-chloride cotransporter 3 interacts with Vav2 to synchronize the cell volume decrease response with cell protrusion dynamicsAdèle Salin-Cantegrel, Masoud Shekarabi, Sarah Rasheed, et al.The Journal of Physiology|April 20, 2004
The human tumour suppressor gene SLC5A8 expresses a Na+-monocarboxylate cotransporterMichael J Coady, Min-Hwang Chang, Francois M Charron, et al.American Journal of Physiology. Renal Physiology|May 22, 2009
Characterization of D150E and G196D aquaporin-2 mutations responsible for nephrogenic diabetes insipidus: importance of a mild phenotypeCécile Guyon, Yoann Lussier, Pierre Bissonnette, et al.Journal of the American Society of Nephrology : JASN|June 12, 2016
MAP17 Is a Necessary Activator of Renal Na+/Glucose Cotransporter SGLT2Michael J Coady, Abdulah El Tarazi, René Santer, et al.Human Molecular Genetics|June 21, 2008
HMSN/ACC truncation mutations disrupt brain-type creatine kinase-dependant activation of K+/Cl- co-transporter 3Adèle Salin-Cantegrel, Masoud Shekarabi, Sébastien Holbert, et al.The Journal of Biological Chemistry|June 2, 2011
Transit defect of potassium-chloride Co-transporter 3 is a major pathogenic mechanism in hereditary motor and sensory neuropathy with agenesis of the corpus callosumAdèle Salin-Cantegrel, Jean-Baptiste Rivière, Masoud Shekarabi, et al.Pageof 4