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The International Journal of Biochemistry & Cell Biology|September 27, 2005
Ca(2+) and Mg(2+) binding to weak sites of TnC C-domain induces exposure of a large hydrophobic surface that leads to loss of TnC from the thin filamentCarolina A C A Braga, José Renato Pinto, Ana Paula Valente, et al.
Biophysical Journal|August 10, 2004
Reversible aggregation plays a crucial role on the folding landscape of p53 core domainDaniella Ishimaru, Luis M T R Lima, Lenize F Maia, et al.
Sub-Cellular Biochemistry|July 16, 2015
Pressure-Inactivated Virus: A Promising Alternative for Vaccine ProductionJerson L Silva, Shana P C Barroso, Ygara S Mendes, et al.
Biochimica Et Biophysica Acta|May 2, 2002
Pressure induces folding intermediates that are crucial for protein-DNA recognition and virus assemblyJerson L Silva, Andréa C Oliveira, Andre M O Gomes, et al.
European Journal of Medicinal Chemistry|August 28, 2010
Synthesis and anti-prion activity evaluation of aminoquinoline analoguesBruno Macedo, Catherine H Kaschula, Roger Hunter, et al.
Plos One|November 16, 2012
Transient transfection of a wild-type p53 gene triggers resveratrol-induced apoptosis in cancer cellsDanielly Cristiny Ferraz da Costa, Fabiana Alves Casanova, Julia Quarti, et al.
Scientific Reports|September 29, 2019
Increase in fatty acids and flotillins upon resveratrol treatment of human breast cancer cellsLuciana Gomes, Marcos Sorgine, Carlos Luan Alves Passos, et al.
Journal of Molecular Biology|October 8, 2003
Conversion of wild-type p53 core domain into a conformation that mimics a hot-spot mutantDaniella Ishimaru, Lenize F Maia, Larissa M Maiolino, et al.
Biomolecules|April 9, 2020
The Status of p53 Oligomeric and Aggregation States in CancerGuilherme A P de Oliveira, Elaine C Petronilho, Murilo M Pedrote, et al.
The Journal of Biological Chemistry|January 4, 2019
p53 reactivation with induction of massive apoptosis-1 (PRIMA-1) inhibits amyloid aggregation of mutant p53 in cancer cellsLuciana P Rangel, Giulia D S Ferretti, Caroline L Costa, et al.
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