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Jiou Wang

Showing results (31-40 of 68) with videos related to

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Elife|July 3, 2026
Non-canonical amino acid incorporation enables minimally disruptive labeling of stress granule and TDP-43 proteinopathyHao Chen, Haocheng Wang, Yu-Ning Lu, et al.
Nature Communications|September 23, 2022
Intracellular energy controls dynamics of stress-induced ribonucleoprotein granulesTao Wang, Xibin Tian, Han Byeol Kim, et al.
Nucleic Acids Research|June 5, 2020
G-quadruplexes offer a conserved structural motif for NONO recruitment to NEAT1 architectural lncRNAEric A J Simko, Honghe Liu, Tao Zhang, et al.
Plos Genetics|January 24, 2009
An ALS-linked mutant SOD1 produces a locomotor defect associated with aggregation and synaptic dysfunction when expressed in neurons of Caenorhabditis elegansJiou Wang, George W Farr, David H Hall, et al.
Plos Biology|March 11, 2021
MARK2 phosphorylates eIF2α in response to proteotoxic stressYu-Ning Lu, Sarah Kavianpour, Tao Zhang, et al.
Plos Biology|April 4, 2015
Regulation of protein quality control by UBE4B and LSD1 through p53-mediated transcriptionGoran Periz, Jiayin Lu, Tao Zhang, et al.
Discovery Medicine|August 30, 2019
Infection with persister forms of Staphylococcus aureus causes a persistent skin infection with more severe lesions in mice: failure to clear the infection by the current standard of care treatmentRebecca Yee, Yuting Yuan, Wanliang Shi, et al.
Human Molecular Genetics|September 11, 2003
Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common featureJiou Wang, Hilda Slunt, Victoria Gonzales, et al.
Neurobiology of Disease|July 20, 2002
Fibrillar inclusions and motor neuron degeneration in transgenic mice expressing superoxide dismutase 1 with a disrupted copper-binding siteJiou Wang, Guilian Xu, Victoria Gonzales, et al.
Cell Metabolism|February 5, 2021
C9orf72 regulates energy homeostasis by stabilizing mitochondrial complex I assemblyTao Wang, Honghe Liu, Kie Itoh, et al.
Pageof 7

Showing results (31-40 of 68) with videos related to

Sort By:
Pageof 7
Elife|July 3, 2026
Non-canonical amino acid incorporation enables minimally disruptive labeling of stress granule and TDP-43 proteinopathyHao Chen, Haocheng Wang, Yu-Ning Lu, et al.
Nature Communications|September 23, 2022
Intracellular energy controls dynamics of stress-induced ribonucleoprotein granulesTao Wang, Xibin Tian, Han Byeol Kim, et al.
Nucleic Acids Research|June 5, 2020
G-quadruplexes offer a conserved structural motif for NONO recruitment to NEAT1 architectural lncRNAEric A J Simko, Honghe Liu, Tao Zhang, et al.
Plos Genetics|January 24, 2009
An ALS-linked mutant SOD1 produces a locomotor defect associated with aggregation and synaptic dysfunction when expressed in neurons of Caenorhabditis elegansJiou Wang, George W Farr, David H Hall, et al.
Plos Biology|March 11, 2021
MARK2 phosphorylates eIF2α in response to proteotoxic stressYu-Ning Lu, Sarah Kavianpour, Tao Zhang, et al.
Plos Biology|April 4, 2015
Regulation of protein quality control by UBE4B and LSD1 through p53-mediated transcriptionGoran Periz, Jiayin Lu, Tao Zhang, et al.
Discovery Medicine|August 30, 2019
Infection with persister forms of Staphylococcus aureus causes a persistent skin infection with more severe lesions in mice: failure to clear the infection by the current standard of care treatmentRebecca Yee, Yuting Yuan, Wanliang Shi, et al.
Human Molecular Genetics|September 11, 2003
Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common featureJiou Wang, Hilda Slunt, Victoria Gonzales, et al.
Neurobiology of Disease|July 20, 2002
Fibrillar inclusions and motor neuron degeneration in transgenic mice expressing superoxide dismutase 1 with a disrupted copper-binding siteJiou Wang, Guilian Xu, Victoria Gonzales, et al.
Cell Metabolism|February 5, 2021
C9orf72 regulates energy homeostasis by stabilizing mitochondrial complex I assemblyTao Wang, Honghe Liu, Kie Itoh, et al.
Pageof 7