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STAR Protocols|October 11, 2021
Lentiviral-mediated ectopic expression of YAP and TAZ in YAPoff cancer cell linesJoel D Pearson, Rod BremnerMolecular & Cellular Oncology|December 3, 2021
Simplifying cancer: binary pan-cancer superclasses stratified by opposite YAP/TEAD effectsJoel D Pearson, Rod BremnerCancers|January 25, 2025
Neuroendocrine Transformation as a Mechanism of Resistance to Targeted Lung Cancer Therapies: Emerging Mechanisms and Their Therapeutic ImplicationsAsim Joshi, Nivitha Bhaskar, Joel D PearsonThe Biochemical Journal|May 29, 2025
Molecular basis and therapeutic implications of binary YAPOn/YAPOff cancer classesPinky Sharma, Yale S Michaels, Joel D PearsonThe Journal of Biological Chemistry|June 11, 2013
Cleavage of the JunB transcription factor by caspases generates a carboxyl-terminal fragment that inhibits activator protein-1 transcriptional activityJason K H Lee, Joel D Pearson, Brandon E Maser, et al.BMC Biochemistry|April 7, 2009
Alpha-sarcin catalytic activity is not required for cytotoxicitySpencer C Alford, Joel D Pearson, Amanda Carette, et al.BMC Cancer|June 12, 2012
The heat shock protein-90 co-chaperone, Cyclophilin 40, promotes ALK-positive, anaplastic large cell lymphoma viability and its expression is regulated by the NPM-ALK oncoproteinJoel D Pearson, Zubair Mohammed, Julinor T C Bacani, et al.Cancer Research Communications|August 22, 2024
Netrin-1 and UNC5B Cooperate with Integrins to Mediate YAP-Driven CytostasisJoel D Pearson, Katherine Huang, Louis G Dela Pena, et al.International Journal of Clinical and Experimental Pathology|February 18, 2011
NPM-ALK and the JunB transcription factor regulate the expression of cytotoxic molecules in ALK-positive, anaplastic large cell lymphomaJoel D Pearson, Jason K H Lee, Julinor T C Bacani, et al.Journal of Signal Transduction|August 2, 2012
NPM-ALK: The Prototypic Member of a Family of Oncogenic Fusion Tyrosine KinasesJoel D Pearson, Jason K H Lee, Julinor T C Bacani, et al.Pageof 3