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John J Schwarz

Showing results (1-10 of 23) with videos related to

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Genesis (New York, N.Y. : 2000)|August 18, 2005
Generation of conditional Mef2cloxP/loxP mice for temporal- and tissue-specific analysesLinh H Vong, Michael J Ragusa, John J Schwarz
Proceedings of the National Academy of Sciences of the United States of America|October 29, 2008
A p38 MAPK-MEF2C pathway regulates B-cell proliferationDustin Khiem, Jason G Cyster, John J Schwarz, et al.
The Biochemical Journal|February 25, 2012
MEF2 is regulated by CaMKIIδ2 and a HDAC4-HDAC5 heterodimer in vascular smooth muscle cellsRoman Ginnan, Li Yan Sun, John J Schwarz, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|May 9, 2006
MEF2C is required for the normal allocation of cells between the ventricular and sinoatrial precursors of the primary heart fieldLinh Vong, Weizhen Bi, Katharine E O'Connor-Halligan, et al.
Circulation Research|January 31, 2014
Deletion of yes-associated protein (YAP) specifically in cardiac and vascular smooth muscle cells reveals a crucial role for YAP in mouse cardiovascular developmentYong Wang, Guoqing Hu, Fang Liu, et al.
Science Advances|November 7, 2018
Cell chirality regulates intercellular junctions and endothelial permeabilityJie Fan, Poulomi Ray, Yao Wei Lu, et al.
Developmental Cell|April 11, 2007
The transcription factor MEF2C is required for craniofacial developmentMichael P Verzi, Pooja Agarwal, Courtney Brown, et al.
The American Journal of Pathology|April 24, 2012
MEF2C ablation in endothelial cells reduces retinal vessel loss and suppresses pathologic retinal neovascularization in oxygen-induced retinopathyZhenhua Xu, Junsong Gong, Debasish Maiti, et al.
Biomaterials|December 13, 2022
Sox17 mediates adult arterial endothelial cell adaptation to hemodynamicsDiana Kim, Alexander Grath, Yao Wei Lu, et al.
Biorxiv : the Preprint Server for Biology|January 16, 2026
Genetic deletion of cytoglobin exacerbates cardiac hypertrophy and inhibits cardiac fibroblast activation independent of changes in blood pressureLe Gia Cat Pham, Kurrim Gilliard, Frances Jourd'heuil, et al.
Pageof 3

Showing results (1-10 of 23) with videos related to

Sort By:
Pageof 3
Genesis (New York, N.Y. : 2000)|August 18, 2005
Generation of conditional Mef2cloxP/loxP mice for temporal- and tissue-specific analysesLinh H Vong, Michael J Ragusa, John J Schwarz
Proceedings of the National Academy of Sciences of the United States of America|October 29, 2008
A p38 MAPK-MEF2C pathway regulates B-cell proliferationDustin Khiem, Jason G Cyster, John J Schwarz, et al.
The Biochemical Journal|February 25, 2012
MEF2 is regulated by CaMKIIδ2 and a HDAC4-HDAC5 heterodimer in vascular smooth muscle cellsRoman Ginnan, Li Yan Sun, John J Schwarz, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|May 9, 2006
MEF2C is required for the normal allocation of cells between the ventricular and sinoatrial precursors of the primary heart fieldLinh Vong, Weizhen Bi, Katharine E O'Connor-Halligan, et al.
Circulation Research|January 31, 2014
Deletion of yes-associated protein (YAP) specifically in cardiac and vascular smooth muscle cells reveals a crucial role for YAP in mouse cardiovascular developmentYong Wang, Guoqing Hu, Fang Liu, et al.
Science Advances|November 7, 2018
Cell chirality regulates intercellular junctions and endothelial permeabilityJie Fan, Poulomi Ray, Yao Wei Lu, et al.
Developmental Cell|April 11, 2007
The transcription factor MEF2C is required for craniofacial developmentMichael P Verzi, Pooja Agarwal, Courtney Brown, et al.
The American Journal of Pathology|April 24, 2012
MEF2C ablation in endothelial cells reduces retinal vessel loss and suppresses pathologic retinal neovascularization in oxygen-induced retinopathyZhenhua Xu, Junsong Gong, Debasish Maiti, et al.
Biomaterials|December 13, 2022
Sox17 mediates adult arterial endothelial cell adaptation to hemodynamicsDiana Kim, Alexander Grath, Yao Wei Lu, et al.
Biorxiv : the Preprint Server for Biology|January 16, 2026
Genetic deletion of cytoglobin exacerbates cardiac hypertrophy and inhibits cardiac fibroblast activation independent of changes in blood pressureLe Gia Cat Pham, Kurrim Gilliard, Frances Jourd'heuil, et al.
Pageof 3