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John W Kyle

Showing results (1-10 of 20) with videos related to

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Cardiac Electrophysiology Clinics|November 15, 2014
Diseases caused by mutations in Na<sub>v</sub>1.5 interacting proteinsJohn W Kyle, Jonathan C Makielski
Science'S STKE : Signal Transduction Knowledge Environment|May 2, 2002
Do defects in ion channel glycosylation set the stage for lethal cardiac arrhythmias?Harry A Fozzard, John W Kyle
American Journal of Physiology. Heart and Circulatory Physiology|February 28, 2006
Flecainide sensitivity of a Na channel long QT mutation shows an open-channel blocking mechanism for use-dependent blockYujie Zhu, John W Kyle, Peter J Lee
The Journal of Physiological Sciences : JPS|April 3, 2009
Late Na+ current produced by human cardiac Na+ channel isoform Nav1.5 is modulated by its beta1 subunitVictor A Maltsev, John W Kyle, Albertas Undrovinas
American Journal of Physiology. Heart and Circulatory Physiology|June 17, 2008
Molecular identity of the late sodium current in adult dog cardiomyocytes identified by Nav1.5 antisense inhibitionVictor A Maltsev, John W Kyle, Sudhish Mishra, et al.
Circulation Research|June 6, 2015
Mechanisms of sudden cardiac death: oxidants and metabolismKai-Chien Yang, John W Kyle, Jonathan C Makielski, et al.
Biochimica Et Biophysica Acta|November 1, 2011
Structural organization of intercellular channels II. Amino terminal domain of the connexins: sequence, functional roles, and structureEric C Beyer, Gregory M Lipkind, John W Kyle, et al.
Molecular Pharmacology|August 4, 2006
An inner pore residue (Asn406) in the Nav1.5 channel controls slow inactivation and enhances mibefradil block to T-type Ca2+ channel levelsMegan M McNulty, John W Kyle, Gregory M Lipkind, et al.
Journal of Molecular and Cellular Cardiology|November 15, 2002
Gating of the late Na+ channel in normal and failing human myocardiumAlbertas I Undrovinas, Victor A Maltsev, John W Kyle, et al.
Journal of Molecular and Cellular Cardiology|April 6, 2013
Channelopathies from mutations in the cardiac sodium channel protein complexGraham S Adsit, Ravi Vaidyanathan, Carla M Galler, et al.
Pageof 2

Showing results (1-10 of 20) with videos related to

Sort By:
Pageof 2
Cardiac Electrophysiology Clinics|November 15, 2014
Diseases caused by mutations in Na<sub>v</sub>1.5 interacting proteinsJohn W Kyle, Jonathan C Makielski
Science'S STKE : Signal Transduction Knowledge Environment|May 2, 2002
Do defects in ion channel glycosylation set the stage for lethal cardiac arrhythmias?Harry A Fozzard, John W Kyle
American Journal of Physiology. Heart and Circulatory Physiology|February 28, 2006
Flecainide sensitivity of a Na channel long QT mutation shows an open-channel blocking mechanism for use-dependent blockYujie Zhu, John W Kyle, Peter J Lee
The Journal of Physiological Sciences : JPS|April 3, 2009
Late Na+ current produced by human cardiac Na+ channel isoform Nav1.5 is modulated by its beta1 subunitVictor A Maltsev, John W Kyle, Albertas Undrovinas
American Journal of Physiology. Heart and Circulatory Physiology|June 17, 2008
Molecular identity of the late sodium current in adult dog cardiomyocytes identified by Nav1.5 antisense inhibitionVictor A Maltsev, John W Kyle, Sudhish Mishra, et al.
Circulation Research|June 6, 2015
Mechanisms of sudden cardiac death: oxidants and metabolismKai-Chien Yang, John W Kyle, Jonathan C Makielski, et al.
Biochimica Et Biophysica Acta|November 1, 2011
Structural organization of intercellular channels II. Amino terminal domain of the connexins: sequence, functional roles, and structureEric C Beyer, Gregory M Lipkind, John W Kyle, et al.
Molecular Pharmacology|August 4, 2006
An inner pore residue (Asn406) in the Nav1.5 channel controls slow inactivation and enhances mibefradil block to T-type Ca2+ channel levelsMegan M McNulty, John W Kyle, Gregory M Lipkind, et al.
Journal of Molecular and Cellular Cardiology|November 15, 2002
Gating of the late Na+ channel in normal and failing human myocardiumAlbertas I Undrovinas, Victor A Maltsev, John W Kyle, et al.
Journal of Molecular and Cellular Cardiology|April 6, 2013
Channelopathies from mutations in the cardiac sodium channel protein complexGraham S Adsit, Ravi Vaidyanathan, Carla M Galler, et al.
Pageof 2