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Cell Reports|April 18, 2019
Simultaneous Live Imaging of Multiple Endogenous Proteins Reveals a Mechanism for Alzheimer's-Related Plasticity ImpairmentSarah G Cook, Dayton J Goodell, Susana Restrepo, et al.
The Journal of Biological Chemistry|April 15, 2023
Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP modeMatthew E Larsen, Olivia R Buonarati, Hai Qian, et al.
Acta Pharmacologica Sinica|June 21, 2011
CaMKII in cerebral ischemiaSteven J Coultrap, Rebekah S Vest, Nicole M Ashpole, et al.
The Journal of Biological Chemistry|July 20, 2011
Nucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2BHeather O'Leary, Wallace H Liu, Jacki M Rorabaugh, et al.
The Journal of Biological Chemistry|April 1, 2010
CaMKII autonomy is substrate-dependent and further stimulated by Ca2+/calmodulinSteven J Coultrap, Isabelle Buard, Jaqueline R Kulbe, et al.
Plos One|August 23, 2013
Camkii-mediated phosphorylation regulates distributions of Syngap-α1 and -α2 at the postsynaptic densityYijung Yang, Jung-Hwa Tao-Cheng, K Ulrich Bayer, et al.
Cell Reports|June 15, 2017
DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP SpecificDayton J Goodell, Vincent Zaegel, Steven J Coultrap, et al.
The Journal of Biological Chemistry|April 29, 2010
Effective post-insult neuroprotection by a novel Ca(2+)/ calmodulin-dependent protein kinase II (CaMKII) inhibitorRebekah S Vest, Heather O'Leary, Steven J Coultrap, et al.
Molecular Pharmacology|May 30, 2026
The CaMKII D135N mutation blocks kinase activity and reduces GluN2B bindingMatthew E Larsen, C Madison Barker, Raul Satoshi Vargas, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 27, 2006
Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2BK Ulrich Bayer, Eric LeBel, Greg L McDonald, et al.
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