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Jonathan P Ling

Showing results (1-10 of 34) with videos related to

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RNA (New York, N.Y.)|March 28, 2018
HnRNP L represses cryptic exonsSean P McClory, Kristen W Lynch, Jonathan P Ling
Science (New York, N.Y.)|August 8, 2015
TDP-43 repression of nonconserved cryptic exons is compromised in ALS-FTDJonathan P Ling, Olga Pletnikova, Juan C Troncoso, et al.
Molecular Neurodegeneration|May 15, 2026
TDP-43: [GU]-ardian of the transcriptomeIrika R Sinha, Abigail L Atkinson, Katherine E Irwin, et al.
Biorxiv : the Preprint Server for Biology|July 16, 2025
Inhibition of nonsense-mediated decay in TDP-43 deficient neurons reveals novel cryptic exonsIrika R Sinha, Yingzhi Ye, Yini Li, et al.
Biorxiv : the Preprint Server for Biology|August 14, 2023
Multivalent GU-rich oligonucleotides sequester TDP-43 in the nucleus by inducing high molecular weight RNP complexesXi Zhang, Tanuza Das, Tiffany F Chao, et al.
Acta Neuropathologica|October 28, 2016
Depletion of TDP-43 decreases fibril and plaque β-amyloid and exacerbates neurodegeneration in an Alzheimer's mouse modelKatherine D LaClair, Aneesh Donde, Jonathan P Ling, et al.
Cell Reports|September 30, 2016
PTBP1 and PTBP2 Repress Nonconserved Cryptic ExonsJonathan P Ling, Resham Chhabra, Jonathan D Merran, et al.
Acta Neuropathologica|July 24, 2019
Splicing repression is a major function of TDP-43 in motor neuronsAneesh Donde, Mingkuan Sun, Jonathan P Ling, et al.
Iscience|July 11, 2024
Multivalent GU-rich oligonucleotides sequester TDP-43 in the nucleus by inducing high molecular weight RNP complexesXi Zhang, Tanuza Das, Tiffany F Chao, et al.
Biorxiv : the Preprint Server for Biology|October 1, 2025
Symptomatic treatment by a BBB-permeable AAV engineered to restore TDP-43 function slows motor neuron disease and prevents paralysisAswathy Peethambaran Mallika, Jessica G Yu, Opal Sitzman, et al.
Pageof 4

Showing results (1-10 of 34) with videos related to

Sort By:
Pageof 4
RNA (New York, N.Y.)|March 28, 2018
HnRNP L represses cryptic exonsSean P McClory, Kristen W Lynch, Jonathan P Ling
Science (New York, N.Y.)|August 8, 2015
TDP-43 repression of nonconserved cryptic exons is compromised in ALS-FTDJonathan P Ling, Olga Pletnikova, Juan C Troncoso, et al.
Molecular Neurodegeneration|May 15, 2026
TDP-43: [GU]-ardian of the transcriptomeIrika R Sinha, Abigail L Atkinson, Katherine E Irwin, et al.
Biorxiv : the Preprint Server for Biology|July 16, 2025
Inhibition of nonsense-mediated decay in TDP-43 deficient neurons reveals novel cryptic exonsIrika R Sinha, Yingzhi Ye, Yini Li, et al.
Biorxiv : the Preprint Server for Biology|August 14, 2023
Multivalent GU-rich oligonucleotides sequester TDP-43 in the nucleus by inducing high molecular weight RNP complexesXi Zhang, Tanuza Das, Tiffany F Chao, et al.
Acta Neuropathologica|October 28, 2016
Depletion of TDP-43 decreases fibril and plaque β-amyloid and exacerbates neurodegeneration in an Alzheimer's mouse modelKatherine D LaClair, Aneesh Donde, Jonathan P Ling, et al.
Cell Reports|September 30, 2016
PTBP1 and PTBP2 Repress Nonconserved Cryptic ExonsJonathan P Ling, Resham Chhabra, Jonathan D Merran, et al.
Acta Neuropathologica|July 24, 2019
Splicing repression is a major function of TDP-43 in motor neuronsAneesh Donde, Mingkuan Sun, Jonathan P Ling, et al.
Iscience|July 11, 2024
Multivalent GU-rich oligonucleotides sequester TDP-43 in the nucleus by inducing high molecular weight RNP complexesXi Zhang, Tanuza Das, Tiffany F Chao, et al.
Biorxiv : the Preprint Server for Biology|October 1, 2025
Symptomatic treatment by a BBB-permeable AAV engineered to restore TDP-43 function slows motor neuron disease and prevents paralysisAswathy Peethambaran Mallika, Jessica G Yu, Opal Sitzman, et al.
Pageof 4