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Methods (San Diego, Calif.)|January 10, 2019
A humanized yeast system to analyze cleavage of prelamin A by ZMPSTE24Eric D Spear, Rebecca F Alford, Tim D Babatz, et al.
British Journal of Clinical Pharmacology|June 30, 2017
Quantitative disease progression model of α-1 proteinase inhibitor therapy on computed tomography lung density in patients with α-1 antitrypsin deficiencyMichael A Tortorici, James A Rogers, Oliver Vit, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 24, 2022
Abolishing the prelamin A ZMPSTE24 cleavage site leads to progeroid phenotypes with near-normal longevity in miceYuexia Wang, Khurts Shilagardi, Trunee Hsu, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 18, 2002
Zmpste24 deficiency in mice causes spontaneous bone fractures, muscle weakness, and a prelamin A processing defectMartin O Bergo, Bryant Gavino, Jed Ross, et al.
Nature Chemical Biology|July 9, 2013
Mapping the functional yeast ABC transporter interactomeJamie Snider, Asad Hanif, Mid Eum Lee, et al.
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