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Joshua Armenia

Showing results (1-10 of 39) with videos related to

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Frontiers in Oncology|July 12, 2023
Impact of DNA damage repair alterations on prostate cancer progression and metastasisNatalia Lukashchuk, Alan Barnicle, Carrie A Adelman, et al.
Molecular Oncology|June 22, 2019
Methylation-associated miR-193b silencing activates master drivers of aggressive prostate cancerYing Z Mazzu, Yuki Yoshikawa, Subhiksha Nandakumar, et al.
Molecular Oncology|March 26, 2014
p70S6 kinase mediates breast cancer cell survival in response to surgical wound fluid stimulationIlenia Segatto, Stefania Berton, Maura Sonego, et al.
Science Advances|August 26, 2022
Cancer-driving mutations are enriched in genic regions intolerant to germline variationDimitrios Vitsios, Ryan S Dhindsa, Dorota Matelska, et al.
Cancer Research Communications|March 27, 2023
Drug-gene Interaction Screens Coupled to Tumor Data Analyses Identify the Most Clinically Relevant Cancer Vulnerabilities Driving Sensitivity to PARP InhibitionKunzah Jamal, Alessandro Galbiati, Joshua Armenia, et al.
Molecular Oncology|May 10, 2020
Ribonucleotide reductase small subunit M2 is a master driver of aggressive prostate cancerYing Z Mazzu, Joshua Armenia, Subhiksha Nandakumar, et al.
Oncogenesis|March 16, 2018
Expression of lncRNA MIR222HG co-transcribed from the miR-221/222 gene promoter facilitates the development of castration-resistant prostate cancerTong Sun, Shin-Yi Du, Joshua Armenia, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 30, 2015
p27kip1 controls H-Ras/MAPK activation and cell cycle entry via modulation of MT stabilityLinda Fabris, Stefania Berton, Ilenia Pellizzari, et al.
Oncotarget|April 15, 2014
Contact inhibition modulates intracellular levels of miR-223 in a p27kip1-dependent mannerJoshua Armenia, Linda Fabris, Francesca Lovat, et al.
Cell Reports|April 5, 2018
Machine Learning Detects Pan-cancer Ras Pathway Activation in The Cancer Genome AtlasGregory P Way, Francisco Sanchez-Vega, Konnor La, et al.
Pageof 4

Showing results (1-10 of 39) with videos related to

Sort By:
Pageof 4
Frontiers in Oncology|July 12, 2023
Impact of DNA damage repair alterations on prostate cancer progression and metastasisNatalia Lukashchuk, Alan Barnicle, Carrie A Adelman, et al.
Molecular Oncology|June 22, 2019
Methylation-associated miR-193b silencing activates master drivers of aggressive prostate cancerYing Z Mazzu, Yuki Yoshikawa, Subhiksha Nandakumar, et al.
Molecular Oncology|March 26, 2014
p70S6 kinase mediates breast cancer cell survival in response to surgical wound fluid stimulationIlenia Segatto, Stefania Berton, Maura Sonego, et al.
Science Advances|August 26, 2022
Cancer-driving mutations are enriched in genic regions intolerant to germline variationDimitrios Vitsios, Ryan S Dhindsa, Dorota Matelska, et al.
Cancer Research Communications|March 27, 2023
Drug-gene Interaction Screens Coupled to Tumor Data Analyses Identify the Most Clinically Relevant Cancer Vulnerabilities Driving Sensitivity to PARP InhibitionKunzah Jamal, Alessandro Galbiati, Joshua Armenia, et al.
Molecular Oncology|May 10, 2020
Ribonucleotide reductase small subunit M2 is a master driver of aggressive prostate cancerYing Z Mazzu, Joshua Armenia, Subhiksha Nandakumar, et al.
Oncogenesis|March 16, 2018
Expression of lncRNA MIR222HG co-transcribed from the miR-221/222 gene promoter facilitates the development of castration-resistant prostate cancerTong Sun, Shin-Yi Du, Joshua Armenia, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 30, 2015
p27kip1 controls H-Ras/MAPK activation and cell cycle entry via modulation of MT stabilityLinda Fabris, Stefania Berton, Ilenia Pellizzari, et al.
Oncotarget|April 15, 2014
Contact inhibition modulates intracellular levels of miR-223 in a p27kip1-dependent mannerJoshua Armenia, Linda Fabris, Francesca Lovat, et al.
Cell Reports|April 5, 2018
Machine Learning Detects Pan-cancer Ras Pathway Activation in The Cancer Genome AtlasGregory P Way, Francisco Sanchez-Vega, Konnor La, et al.
Pageof 4