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Neuron|September 28, 2010
PolyQ disease: too many Qs, too much function?Ian H Kratter, Steven FinkbeinerNeuron|April 24, 2015
Going retro: ancient viral origins of cognitionMatthew R Campioni, Steven FinkbeinerTrends in Neurosciences|April 10, 2007
NMDA and AMPA receptors: old channels, new tricksVikram R Rao, Steven FinkbeinerCurrent Opinion in Neurobiology|November 6, 2009
High-content screening of primary neurons: ready for prime timeAaron Daub, Punita Sharma, Steven FinkbeinerFaculty Reviews|July 22, 2022
Huntington's disease iPSC models-using human patient cells to understand the pathology caused by expanded CAG repeatsJulia Kaye, Terry Reisine, Steven FinkbeinerExpert Opinion on Drug Discovery|June 11, 2019
Cell death assays for neurodegenerative disease drug discoveryJeremy W Linsley, Terry Reisine, Steven FinkbeinerNeuron|April 10, 2015
Cell-based screening: extracting meaning from complex dataSteven Finkbeiner, Michael Frumkin, Paul D KassnerAutophagy|October 20, 2009
Protein turnover and inclusion body formationSiddhartha Mitra, Andrey S Tsvetkov, Steven FinkbeinerThe Journal of Biological Chemistry|December 17, 2008
Single neuron ubiquitin-proteasome dynamics accompanying inclusion body formation in huntington diseaseSiddhartha Mitra, Andrey S Tsvetkov, Steven FinkbeinerThe Journal of Biological Chemistry|June 2, 2024
Three dimensional and four dimensional live imaging to study mechanisms of progressive neurodegenerationJeremy W Linsley, Terry Reisine, Steven FinkbeinerPageof 14