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Molecular Pharmacology|August 17, 2018
Compensatory Expression of Nur77 and Nurr1 Regulates NF-κB-Dependent Inflammatory Signaling in AstrocytesKatriana A Popichak, Sean L Hammond, Julie A Moreno, et al.
Frontiers in Neuroscience|June 5, 2023
Gliosis, misfolded protein aggregation, and neuronal loss in a guinea pig model of pulmonary tuberculosisAmanda S Latham, Charlize E Geer, David F Ackart, et al.
Plos One|December 31, 2024
Immune cell infiltration and modulation of the blood-brain barrier in a guinea pig model of tuberculosis: Observations without evidence of bacterial dissemination to the brainAmanda S Latham, Charlize E Geer, David F Ackart, et al.
Journal of Neuroinflammation|July 27, 2024
Nanoligomers targeting NF-κB and NLRP3 reduce neuroinflammation and improve cognitive function with aging and tauopathyDevin Wahl, Sydney J Risen, Shelby C Osburn, et al.
Biorxiv : the Preprint Server for Biology|February 19, 2024
Nanoligomers targeting NF-κB and NLRP3 reduce neuroinflammation and improve cognitive function with aging and tauopathyDevin Wahl, Sydney J Risen, Shelby C Osburn, et al.
Journal of Leukocyte Biology|March 4, 2025
Suppression of NF-κB/NLRP3 by nanoligomer therapy mitigates ethanol and advanced age-related neuroinflammationPaige E Anton, Shannon Twardy, Prashant Nagpal, et al.
Acta Physiologica (Oxford, England)|June 11, 2024
Non-transgenic guinea pig strains exhibit divergent age-related changes in hippocampal mitochondrial respirationMaureen A Walsh, Amanda S Latham, Qian Zhang, et al.
Frontiers in Neuroscience|May 30, 2023
Intranasally delivered mesenchymal stromal cells decrease glial inflammation early in prion diseaseArielle J D Hay, Amanda S Latham, Genova Mumford, et al.
The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences|March 24, 2022
Nontransgenic Guinea Pig Strains Exhibit Hallmarks of Human Brain Aging and Alzheimer's DiseaseDevin Wahl, Julie A Moreno, Kelly S Santangelo, et al.
Journal of Immunology (Baltimore, Md. : 1950)|October 27, 2017
Complement Regulatory Protein Factor H Is a Soluble Prion Receptor That Potentiates Peripheral Prion PathogenesisSarah J Kane, Taylor K Farley, Elizabeth O Gordon, et al.
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