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Justus B Cohen

Showing results (11-20 of 47) with videos related to

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Expert Review of Neurotherapeutics|April 7, 2009
Design and application of oncolytic HSV vectors for glioblastoma therapyPaola Grandi, Pierpaolo Peruzzi, Bonnie Reinhart, et al.
Methods in Molecular Biology (Clifton, N.J.)|October 17, 2019
Engineering HSV-1 Vectors for Gene TherapyWilliam F Goins, Shaohua Huang, Bonnie Hall, et al.
Molecular Therapy. Methods & Clinical Development|July 31, 2023
Novel mutations in U<sub>L</sub>24 and gH rescue efficient infection of an HSV vector retargeted to TrkAMarco Marzulli, Bonnie L Hall, Mingdi Zhang, et al.
International Journal of Molecular Sciences|November 25, 2020
Generation of an Oncolytic Herpes Simplex Viral Vector Completely Retargeted to the GDNF Receptor GFRα1 for Specific Infection of Breast Cancer CellsBonnie L Hall, Daniela Leronni, Yoshitaka Miyagawa, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|April 4, 2025
Next-generation replication-defective HSV vectors for delivery of large DNA payloadsSelene Ingusci, William F Goins, Justus B Cohen, et al.
Journal of Virology|November 16, 2012
Novel mutations in gB and gH circumvent the requirement for known gD Receptors in herpes simplex virus 1 entry and cell-to-cell spreadHiroaki Uchida, Janet Chan, Indira Shrivastava, et al.
Current Cancer Drug Targets|February 9, 2017
Oncolytic Herpes Simplex Virus Vectors Fully Retargeted to Tumor- Associated AntigensHiroaki Uchida, Hirofumi Hamada, Kenji Nakano, et al.
Journal of Virology|September 24, 2010
A double mutation in glycoprotein gB compensates for ineffective gD-dependent initiation of herpes simplex virus type 1 infectionHiroaki Uchida, Janet Chan, William F Goins, et al.
Virology|March 7, 2008
Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growthHan Li, Rajasekaran Baskaran, David M Krisky, et al.
Journal of Virology|August 3, 2007
Equine herpesvirus 1 enters cells by two different pathways, and infection requires the activation of the cellular kinase ROCK1Arthur R Frampton, Donna B Stolz, Hiroaki Uchida, et al.
Pageof 5

Showing results (11-20 of 47) with videos related to

Sort By:
Pageof 5
Expert Review of Neurotherapeutics|April 7, 2009
Design and application of oncolytic HSV vectors for glioblastoma therapyPaola Grandi, Pierpaolo Peruzzi, Bonnie Reinhart, et al.
Methods in Molecular Biology (Clifton, N.J.)|October 17, 2019
Engineering HSV-1 Vectors for Gene TherapyWilliam F Goins, Shaohua Huang, Bonnie Hall, et al.
Molecular Therapy. Methods & Clinical Development|July 31, 2023
Novel mutations in U<sub>L</sub>24 and gH rescue efficient infection of an HSV vector retargeted to TrkAMarco Marzulli, Bonnie L Hall, Mingdi Zhang, et al.
International Journal of Molecular Sciences|November 25, 2020
Generation of an Oncolytic Herpes Simplex Viral Vector Completely Retargeted to the GDNF Receptor GFRα1 for Specific Infection of Breast Cancer CellsBonnie L Hall, Daniela Leronni, Yoshitaka Miyagawa, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|April 4, 2025
Next-generation replication-defective HSV vectors for delivery of large DNA payloadsSelene Ingusci, William F Goins, Justus B Cohen, et al.
Journal of Virology|November 16, 2012
Novel mutations in gB and gH circumvent the requirement for known gD Receptors in herpes simplex virus 1 entry and cell-to-cell spreadHiroaki Uchida, Janet Chan, Indira Shrivastava, et al.
Current Cancer Drug Targets|February 9, 2017
Oncolytic Herpes Simplex Virus Vectors Fully Retargeted to Tumor- Associated AntigensHiroaki Uchida, Hirofumi Hamada, Kenji Nakano, et al.
Journal of Virology|September 24, 2010
A double mutation in glycoprotein gB compensates for ineffective gD-dependent initiation of herpes simplex virus type 1 infectionHiroaki Uchida, Janet Chan, William F Goins, et al.
Virology|March 7, 2008
Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growthHan Li, Rajasekaran Baskaran, David M Krisky, et al.
Journal of Virology|August 3, 2007
Equine herpesvirus 1 enters cells by two different pathways, and infection requires the activation of the cellular kinase ROCK1Arthur R Frampton, Donna B Stolz, Hiroaki Uchida, et al.
Pageof 5