K F Sheu|C W Hu|M F Utter - JoVE Visualize

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Archives of Neurology|August 1, 1988

Reduced activities of thiamine-dependent enzymes in the brains and peripheral tissues of patients with Alzheimer's diseaseG E Gibson, K F Sheu, J P Blass, et al.

Pediatric Research|May 1, 1983

Hypoglycemia, hepatic dysfunction, muscle weakness, cardiomyopathy, free carnitine deficiency and long-chain acylcarnitine excess responsive to medium chain triglyceride dietA M Glasgow, A G Engel, D M Bier, et al.

Journal of Neurochemistry|February 26, 1999

Pathogenesis of inclusion bodies in (CAG)n/Qn-expansion diseases with special reference to the role of tissue transglutaminase and to selective vulnerabilityA J Cooper, K F Sheu, J R Burke, et al.

Neurology|May 5, 1999

A DLST genotype associated with reduced risk for Alzheimer's diseaseK F Sheu, A M Brown, B S Kristal, et al.

Pediatrics|December 1, 1984

Central hypoventilation syndrome in pyruvate dehydrogenase complex deficiencyK Johnston, C J Newth, K F Sheu, et al.

Journal of Neurochemistry|April 27, 1999

Metabolic impairment induces oxidative stress, compromises inflammatory responses, and inactivates a key mitochondrial enzyme in microgliaL C Park, H Zhang, K F Sheu, et al.

Experimental Neurology|February 23, 2000

Neuronal subclass-selective loss of pyruvate dehydrogenase immunoreactivity following canine cardiac arrest and resuscitationY E Bogaert, K F Sheu, P R Hof, et al.

Cellular Immunology|April 15, 1988

Studies on suppressor factors produced by T-cell hybridomas. I. Characterization of antigen-specific suppressor factorsY T Kim, K F Sheu, J J Gibbons, et al.

Brain Research|April 17, 1995

Accumulation of amyloid precursor protein-like immunoreactivity in rat brain in response to thiamine deficiencyN Y Calingasan, S E Gandy, H Baker, et al.

Age|April 23, 2013

Inhibition of α-ketoglutarate-and pyruvate dehydrogenase complexes in E. coli by a glutathione S-transferase containing a pathological length poly-Q domain: A possible role of energy deficit in neurological diseases associated with poly-Q expansions?A J Cooper, K F Sheu, J R Burke, et al.

Pageof 11

Showing results (71-80 of 108) with videos related to

Sort By:

Pageof 11

Archives of Neurology|August 1, 1988

Reduced activities of thiamine-dependent enzymes in the brains and peripheral tissues of patients with Alzheimer's diseaseG E Gibson, K F Sheu, J P Blass, et al.

Pediatric Research|May 1, 1983

Hypoglycemia, hepatic dysfunction, muscle weakness, cardiomyopathy, free carnitine deficiency and long-chain acylcarnitine excess responsive to medium chain triglyceride dietA M Glasgow, A G Engel, D M Bier, et al.

Journal of Neurochemistry|February 26, 1999

Pathogenesis of inclusion bodies in (CAG)n/Qn-expansion diseases with special reference to the role of tissue transglutaminase and to selective vulnerabilityA J Cooper, K F Sheu, J R Burke, et al.

Neurology|May 5, 1999

A DLST genotype associated with reduced risk for Alzheimer's diseaseK F Sheu, A M Brown, B S Kristal, et al.

Pediatrics|December 1, 1984

Central hypoventilation syndrome in pyruvate dehydrogenase complex deficiencyK Johnston, C J Newth, K F Sheu, et al.

Journal of Neurochemistry|April 27, 1999

Metabolic impairment induces oxidative stress, compromises inflammatory responses, and inactivates a key mitochondrial enzyme in microgliaL C Park, H Zhang, K F Sheu, et al.

Experimental Neurology|February 23, 2000

Neuronal subclass-selective loss of pyruvate dehydrogenase immunoreactivity following canine cardiac arrest and resuscitationY E Bogaert, K F Sheu, P R Hof, et al.

Cellular Immunology|April 15, 1988

Studies on suppressor factors produced by T-cell hybridomas. I. Characterization of antigen-specific suppressor factorsY T Kim, K F Sheu, J J Gibbons, et al.

Brain Research|April 17, 1995

Accumulation of amyloid precursor protein-like immunoreactivity in rat brain in response to thiamine deficiencyN Y Calingasan, S E Gandy, H Baker, et al.

Age|April 23, 2013

Inhibition of α-ketoglutarate-and pyruvate dehydrogenase complexes in E. coli by a glutathione S-transferase containing a pathological length poly-Q domain: A possible role of energy deficit in neurological diseases associated with poly-Q expansions?A J Cooper, K F Sheu, J R Burke, et al.

Pageof 11