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European Heart Journal|June 20, 2012
The tryptophan metabolite 3-hydroxyanthranilic acid lowers plasma lipids and decreases atherosclerosis in hypercholesterolaemic miceLei Zhang, Olga Ovchinnikova, Andreas Jönsson, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|October 29, 2005
Expression of neutrophil gelatinase-associated lipocalin in atherosclerosis and myocardial infarctionAnne-Louise Hemdahl, Anders Gabrielsen, Chaoyong Zhu, et al.Molecular Medicine (Cambridge, Mass.)|February 29, 2012
Prediction of ischemic events on the basis of transcriptomic and genomic profiling in patients undergoing carotid endarterectomyLasse Folkersen, Jonas Persson, Johan Ekstrand, et al.Cardiovascular Drugs and Therapy|March 15, 2006
Gene deletion of NF-kappaB p105 enhances neointima formation in a mouse model of carotid artery injuryArno Ruusalepp, Zhong-Qun Yan, Harald Carlsen, et al.CPT: Pharmacometrics & Systems Pharmacology|December 6, 2013
PKPD Modeling of Predictors for Adverse Effects and Overall Survival in Sunitinib-Treated Patients With GISTE K Hansson, G Ma, M A Amantea, et al.Journal of Molecular and Cellular Cardiology|March 9, 2007
Gene expression signals involved in ischemic injury, extracellular matrix composition and fibrosis defined by global mRNA profiling of the human left ventricular myocardiumAnders Gabrielsen, Patrick R Lawler, Wang Yongzhong, et al.Circulation|August 28, 2002
Accumulation of foam cells in liver X receptor-deficient miceGertrud U Schuster, Paolo Parini, Ling Wang, et al.Circulation|March 16, 2011
Upregulation of the 5-lipoxygenase pathway in human aortic valves correlates with severity of stenosis and leads to leukotriene-induced effects on valvular myofibroblastsEdit Nagy, Daniel C Andersson, Kenneth Caidahl, et al.Nature Reviews. Disease Primers|August 18, 2019
AtherosclerosisPeter Libby, Julie E Buring, Lina Badimon, et al.Thrombosis and Haemostasis|May 13, 2014
Apolipoprotein B100 danger-associated signal 1 (ApoBDS-1) triggers platelet activation and boosts platelet-leukocyte proinflammatory responsesA Assinger, Y Wang, L M Butler, et al.Pageof 39