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European Journal of Vascular and Endovascular Surgery : the Official Journal of the European Society for Vascular Surgery|November 6, 2009
VEGF: a surrogate marker for peripheral vascular diseaseA Stehr, I Töpel, S Müller, et al.
Anticancer Research|July 4, 2012
Targeting HSP90 by the novel inhibitor NVP-AUY922 reduces growth and angiogenesis of pancreatic cancerChristian Moser, Sven A Lang, Christina Hackl, et al.
Haemostasis|January 1, 1990
1-Deamino-8-D-arginine vasopressin in the treatment of non-haemophilic patients with acquired factor VIII inhibitorM Muhm, N Grois, P Kier, et al.
Clinical Cardiology|March 1, 1991
Pacing-induced myocardial ischemia does not affect the endothelial release of coagulant and fibrinolytic factors into the coronary circulationH D Gössinger, W Speiser, P Siostrzonek, et al.
Human Pathology|December 31, 1997
Expression of fibrinolytic antigens in redistributed cardiac mast cells in auricular thrombosisH C Bankl, T Radaszkiewicz, B Pikula, et al.
Acta Haematologica|January 1, 1982
Systemic candidiasis complicating bone marrow transplantation in aplastic anemia. Case reportW Hinterberger, L Fridrich, W Graninger, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|March 1, 1990
Prognostic significance of surface marker expression on blasts of patients with de novo acute myeloblastic leukemiaI Schwarzinger, P Valent, U Köller, et al.
British Journal of Haematology|March 1, 1985
Cell lineage heterogeneity in blast crisis of chronic myeloid leukaemiaP Bettelheim, D Lutz, O Majdic, et al.
The Journal of Clinical Investigation|June 1, 1985
Deficiency of the autologous mixed lymphocyte reaction in patients with classic hemophilia treated with commercial factor VIII concentrate. Correlation with T cell subset distribution, antibodies to lymphadenopathy-associated or human T lymphotropic virus, and analysis of the cellular basis of the deficiencyJ S Smolen, P Bettelheim, U Köller, et al.
British Journal of Haematology|December 31, 1997
A hitherto unknown splice site defect in the protein S gene (PROS1): the mutation results in allelic exclusion and causes type I and type III protein S deficiencyS Mustafa, I Pabinger, K Vàradi, et al.
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