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Medical Hypotheses|November 1, 2003
An interaction between type 1 and type 2 programmed cell death and clonogenic survivalK M Anderson, P Bonomi, Y Hu, et al.Medical Hypotheses|July 1, 1996
Does an inability to eradicate normal stem cells preclude the cure of some cancers?K M Anderson, P Bonomi, J E HarrisMedical Hypotheses|October 1, 1994
Potential applications of apoptosis in modifying the biological behavior of therapeutically refractory cancersK M Anderson, J E Harris, P BonomiMedical Hypotheses|May 8, 1998
Inhibition of cancer cell proliferation by disruption of interdigitated/concatenated hierarchies of metabolic control/implementation processes: a proposalK M Anderson, P Bonomi, J Meng, et al.Medical Hypotheses|May 26, 1999
Free radical spin traps as adjuncts for the prevention and treatment of diseaseK M Anderson, G Ells, P Bonomi, et al.Medical Hypotheses|June 11, 1999
Does maximizing programmed cell death necessarily yield an optimum clinical advantage?D Ou, K M Anderson, W Leslie, et al.Prostaglandins, Leukotrienes, and Essential Fatty Acids|June 11, 2002
Increased cytosol Ca(2+) and type 1 programmed cell death in Bcl-2-positive U937 but not in Bcl-2-negative PC-3 and Panc-1 cells induced by the 5-lipoxygenase inhibitor MK 886K M Anderson, W A Alrefai, P K Dudeja, et al.Cancer Letters|April 20, 2001
The mode of cell death in H-358 lung cancer cells cultured with inhibitors of 5-lipoxygenase or the free radical spin trap, NTBND Ou, P Bonomi, W Jao, et al.Medical Hypotheses|June 27, 2000
Widespread countervailing genomic responses induced by chemotherapy or radiation as a cause of therapeutic failureK M Anderson, W A Alrefai, C A Anderson, et al.Clinical Physiology and Biochemistry|January 1, 1985
Metastatic breast cancer from an occult primary provisionally identified by steroid receptor content and two-dimensional protein electrophoresis: some problems associated with the use of the latter techniqueK M Anderson, P BonomiPageof 358