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Antioxidants & Redox Signaling|November 1, 2003
The role of endogenous NADPH oxidases in airway and pulmonary vascular smooth muscle functionJohn R Hoidal, S S Brar, Anne B Sturrock, et al.
Free Radical Biology & Medicine|December 31, 2022
The receptor for advanced glycation end products (RAGE) is involved in mitochondrial function and cigarette smoke-induced oxidative stressOh Sung Kwon, Stephen T Decker, Jia Zhao, et al.
American Journal of Physiology. Endocrinology and Metabolism|June 14, 2021
Skeletal muscle mitochondrial adaptations induced by long-term cigarette smoke exposureStephen T Decker, Oh-Sung Kwon, Jia Zhao, et al.
Scientific Reports|January 20, 2019
RAGE is a Critical Mediator of Pulmonary Oxidative Stress, Alveolar Macrophage Activation and Emphysema in Response to Cigarette SmokeKarl A Sanders, Don A Delker, Tom Huecksteadt, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|April 23, 2024
Progesterone amplifies allergic inflammation and airway pathology in association with higher lung ILC2 responsesShubhanshi Trivedi, Cassandra E Deering-Rice, Samuel E Aamodt, et al.
Frontiers in Immunology|August 18, 2025
Serum 17<i>β</i>-estradiol inversely correlates with circulating group 2 innate lymphoid cells in a cohort of asthmatic patientsElizabeth J Myers, Samuel E Aamodt, Thomas P Huecksteadt, et al.
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