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Kathleen M Mulvaney

Showing results (1-10 of 13) with videos related to

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Cancer Discovery|November 1, 2023
Early Clinical Success of MTA-Cooperative PRMT5 Inhibitors for the Treatment of CDKN2A/MTAP-Deleted CancersKathleen M Mulvaney
Cancer Research|July 2, 2026
ARF beyond p53: Shaping the Pancreatic Tumor MicroenvironmentEliana Destefanis, Kathleen M Mulvaney
Current Opinion in Toxicology|August 1, 2025
Dissecting the Keap1/Nrf2 pathway through proteomicsTigist Y Tamir, Kathleen M Mulvaney, M Ben Major
The Journal of Biological Chemistry|January 10, 2025
Substrate adaptors are flexible tethering modules that enhance substrate methylation by the arginine methyltransferase PRMT5Cyrus Y Jin, Moritz Hunkeler, Kathleen M Mulvaney, et al.
Protein Engineering, Design & Selection : PEDS|October 23, 2015
Engineering a genetically encoded competitive inhibitor of the KEAP1-NRF2 interaction via structure-based design and phage displayGurkan Guntas, Steven M Lewis, Kathleen M Mulvaney, et al.
Environment International|February 2, 2020
Health co-benefits and mitigation costs as per the Paris Agreement under different technological pathways for energy supplyJon Sampedro, Steven J Smith, Iñaki Arto, et al.
Cancer Research|February 6, 2013
Proteomic analysis of ubiquitin ligase KEAP1 reveals associated proteins that inhibit NRF2 ubiquitinationBridgid E Hast, Dennis Goldfarb, Kathleen M Mulvaney, et al.
Environmental Science & Technology|January 4, 2020
Mercury Benefits of Climate Policy in China: Addressing the Paris Agreement and the Minamata Convention SimultaneouslyKathleen M Mulvaney, Noelle E Selin, Amanda Giang, et al.
The Journal of Biological Chemistry|September 14, 2016
Identification and Characterization of MCM3 as a Kelch-like ECH-associated Protein 1 (KEAP1) SubstrateKathleen M Mulvaney, Jacob P Matson, Priscila F Siesser, et al.
The Journal of Pathology|July 4, 2020
A conditional mouse expressing an activating mutation in NRF2 displays hyperplasia of the upper gastrointestinal tract and decreased white adipose tissueBrittany M Bowman, Stephanie A Montgomery, Travis P Schrank, et al.
Pageof 2

Showing results (1-10 of 13) with videos related to

Sort By:
Pageof 2
Cancer Discovery|November 1, 2023
Early Clinical Success of MTA-Cooperative PRMT5 Inhibitors for the Treatment of CDKN2A/MTAP-Deleted CancersKathleen M Mulvaney
Cancer Research|July 2, 2026
ARF beyond p53: Shaping the Pancreatic Tumor MicroenvironmentEliana Destefanis, Kathleen M Mulvaney
Current Opinion in Toxicology|August 1, 2025
Dissecting the Keap1/Nrf2 pathway through proteomicsTigist Y Tamir, Kathleen M Mulvaney, M Ben Major
The Journal of Biological Chemistry|January 10, 2025
Substrate adaptors are flexible tethering modules that enhance substrate methylation by the arginine methyltransferase PRMT5Cyrus Y Jin, Moritz Hunkeler, Kathleen M Mulvaney, et al.
Protein Engineering, Design & Selection : PEDS|October 23, 2015
Engineering a genetically encoded competitive inhibitor of the KEAP1-NRF2 interaction via structure-based design and phage displayGurkan Guntas, Steven M Lewis, Kathleen M Mulvaney, et al.
Environment International|February 2, 2020
Health co-benefits and mitigation costs as per the Paris Agreement under different technological pathways for energy supplyJon Sampedro, Steven J Smith, Iñaki Arto, et al.
Cancer Research|February 6, 2013
Proteomic analysis of ubiquitin ligase KEAP1 reveals associated proteins that inhibit NRF2 ubiquitinationBridgid E Hast, Dennis Goldfarb, Kathleen M Mulvaney, et al.
Environmental Science & Technology|January 4, 2020
Mercury Benefits of Climate Policy in China: Addressing the Paris Agreement and the Minamata Convention SimultaneouslyKathleen M Mulvaney, Noelle E Selin, Amanda Giang, et al.
The Journal of Biological Chemistry|September 14, 2016
Identification and Characterization of MCM3 as a Kelch-like ECH-associated Protein 1 (KEAP1) SubstrateKathleen M Mulvaney, Jacob P Matson, Priscila F Siesser, et al.
The Journal of Pathology|July 4, 2020
A conditional mouse expressing an activating mutation in NRF2 displays hyperplasia of the upper gastrointestinal tract and decreased white adipose tissueBrittany M Bowman, Stephanie A Montgomery, Travis P Schrank, et al.
Pageof 2