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Shock (Augusta, Ga.)|July 4, 2007
Inhibition of L-type calcium channels in arteriolar smooth muscle cells is involved in the pathogenesis of vascular hyporeactivity in severe shockQing Zhao, Ke-seng ZhaoWorld Journal of Gastroenterology|January 31, 2002
Studies on the flow and distribution of leukocytes in mesentery microcirculation of ratsYong Jiang, Ai-Hua Liu, Ke-Seng ZhaoJournal of Applied Toxicology : JAT|May 21, 2011
Atractyloside induces low contractile reaction of arteriolar smooth muscle through mitochondrial damageRui Song, Huining Bian, Xuliang Huang, et al.American Journal of Hypertension|October 14, 2010
Mitochondrial injury underlies hyporeactivity of arterial smooth muscle in severe shockRui Song, Huining Bian, Xingmin Wang, et al.Expert Opinion on Investigational Drugs|December 18, 2012
Polydatin--a new mitochondria protector for acute severe hemorrhagic shock treatmentXingmin Wang, Rui Song, Yunyan Chen, et al.Expert Opinion on Therapeutic Targets|June 16, 2015
Polydatin protects hepatocytes against mitochondrial injury in acute severe hemorrhagic shock via SIRT1-SOD2 pathwayPengyun Li, Xingmin Wang, Ming Zhao, et al.International Journal of Molecular Medicine|March 25, 2017
SIRT1 is required for mitochondrial biogenesis reprogramming in hypoxic human pulmonary arteriolar smooth muscle cellsPengyun Li, Yan Liu, Nana Burns, et al.The Journal of Biological Chemistry|April 6, 2017
An epigenetic regulatory loop controls pro-osteogenic activation by TGF-β1 or bone morphogenetic protein 2 in human aortic valve interstitial cellsRui Song, David A Fullerton, Lihua Ao, et al.Redox Report : Communications in Free Radical Research|March 24, 2004
Mobilization of intracellular calcium by peroxynitrite in arteriolar smooth muscle cells from ratsBing-Xing Pan, Gui-Ling Zhao, Xu-Liang Huang, et al.Pageof 4