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Nature Communications|February 19, 2025
A decision point between transdifferentiation and programmed cell death priming controls KRAS-dependent pancreatic cancer developmentAnne T Schneider, Christiane Koppe, Emilie Crouchet, et al.Cancer Cell|June 14, 2017
Kupffer Cell-Derived Tnf Triggers Cholangiocellular Tumorigenesis through JNK due to Chronic Mitochondrial Dysfunction and ROSDetian Yuan, Shan Huang, Emanuel Berger, et al.Nature Communications|March 31, 2019
PiggyBac transposon tools for recessive screening identify B-cell lymphoma drivers in miceJulia Weber, Jorge de la Rosa, Carolyn S Grove, et al.Nature|January 25, 2018
Evolutionary routes and KRAS dosage define pancreatic cancer phenotypesSebastian Mueller, Thomas Engleitner, Roman Maresch, et al.Cancer Cell|September 13, 2017
A Dual Role of Caspase-8 in Triggering and Sensing Proliferation-Associated DNA Damage, a Key Determinant of Liver Cancer DevelopmentYannick Boege, Mohsen Malehmir, Marc E Healy, et al.Nature Medicine|April 3, 2019
Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancerMohsen Malehmir, Dominik Pfister, Suchira Gallage, et al.Immunity|June 17, 2023
Sublethal necroptosis signaling promotes inflammation and liver cancerMihael Vucur, Ahmed Ghallab, Anne T Schneider, et al.Journal of Hepatology|May 24, 2023
Intestinal B cells license metabolic T-cell activation in NASH microbiota/antigen-independently and contribute to fibrosis by IgA-FcR signallingElena Kotsiliti, Valentina Leone, Svenja Schuehle, et al.Nature|February 4, 2026
Activated ATF6α is a hepatic tumour driver restricting immunosurveillanceXin Li, Cynthia Lebeaupin, Aikaterini Kadianaki, et al.Nature|March 25, 2021
NASH limits anti-tumour surveillance in immunotherapy-treated HCCDominik Pfister, Nicolás Gonzalo Núñez, Roser Pinyol, et al.Pageof 13