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L A Hansen

Showing results (91-100 of 140) with videos related to

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Brain Research|February 21, 1992
Casein kinase II is associated with neurofibrillary tangles but is not an intrinsic component of paired helical filamentsL Baum, E Masliah, D S Iimoto, et al.
Cancer Research|July 26, 2000
The epidermal growth factor receptor is required to maintain the proliferative population in the basal compartment of epidermal tumorsL A Hansen, R L Woodson, S Holbus, et al.
Science (New York, N.Y.)|April 3, 2001
Conservation conflicts across AfricaA Balmford, J L Moore, T Brooks, et al.
Neuroscience|January 1, 1989
Substance P and somatostatin coexist within neuritic plaques: implications for the pathogenesis of Alzheimer's diseaseD M Armstrong, W C Benzing, J Evans, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|June 18, 2003
Early and persistent alterations in prefrontal cortex MAO A and B in Alzheimer's diseaseB P Kennedy, M G Ziegler, M Alford, et al.
Neurology|March 1, 1995
The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part X. Neuropathology confirmation of the clinical diagnosis of Alzheimer's diseaseM Gearing, S S Mirra, J C Hedreen, et al.
Archives of Neurology|July 1, 1995
Cerebral infarction in Alzheimer's disease is associated with severe amyloid angiopathy and hypertensionJ M Olichney, L A Hansen, C R Hofstetter, et al.
Annals of the New York Academy of Sciences|May 20, 2000
Relationship between severe amyloid angiopathy, apolipoprotein E genotype, and vascular lesions in Alzheimer's diseaseJ M Olichney, L A Hansen, J H Lee, et al.
Brain and Cognition|July 1, 1996
Neuropsychological deficits associated with diffuse Lewy body diseaseD P Salmon, D Galasko, L A Hansen, et al.
Neurology|January 10, 2001
Altered expression of synaptic proteins occurs early during progression of Alzheimer's diseaseE Masliah, M Mallory, M Alford, et al.
Pageof 14

Showing results (91-100 of 140) with videos related to

Sort By:
Pageof 14
Brain Research|February 21, 1992
Casein kinase II is associated with neurofibrillary tangles but is not an intrinsic component of paired helical filamentsL Baum, E Masliah, D S Iimoto, et al.
Cancer Research|July 26, 2000
The epidermal growth factor receptor is required to maintain the proliferative population in the basal compartment of epidermal tumorsL A Hansen, R L Woodson, S Holbus, et al.
Science (New York, N.Y.)|April 3, 2001
Conservation conflicts across AfricaA Balmford, J L Moore, T Brooks, et al.
Neuroscience|January 1, 1989
Substance P and somatostatin coexist within neuritic plaques: implications for the pathogenesis of Alzheimer's diseaseD M Armstrong, W C Benzing, J Evans, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|June 18, 2003
Early and persistent alterations in prefrontal cortex MAO A and B in Alzheimer's diseaseB P Kennedy, M G Ziegler, M Alford, et al.
Neurology|March 1, 1995
The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part X. Neuropathology confirmation of the clinical diagnosis of Alzheimer's diseaseM Gearing, S S Mirra, J C Hedreen, et al.
Archives of Neurology|July 1, 1995
Cerebral infarction in Alzheimer's disease is associated with severe amyloid angiopathy and hypertensionJ M Olichney, L A Hansen, C R Hofstetter, et al.
Annals of the New York Academy of Sciences|May 20, 2000
Relationship between severe amyloid angiopathy, apolipoprotein E genotype, and vascular lesions in Alzheimer's diseaseJ M Olichney, L A Hansen, J H Lee, et al.
Brain and Cognition|July 1, 1996
Neuropsychological deficits associated with diffuse Lewy body diseaseD P Salmon, D Galasko, L A Hansen, et al.
Neurology|January 10, 2001
Altered expression of synaptic proteins occurs early during progression of Alzheimer's diseaseE Masliah, M Mallory, M Alford, et al.
Pageof 14