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Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|February 29, 1996
The regulation of AP-1 activity by mitogen-activated protein kinasesM KarinThe New Biologist|February 1, 1990
Too many transcription factors: positive and negative interactionsM KarinThe Journal of Clinical Investigation|July 4, 2001
c-Jun N-terminal kinase is required for metalloproteinase expression and joint destruction in inflammatory arthritisZ Han, D L Boyle, L Chang, et al.Immunity|January 8, 2000
JNK2 and IKKbeta are required for activating the innate response to viral infectionW M Chu, D Ostertag, Z W Li, et al.Cell Death and Differentiation|September 6, 2014
Roles of tumor suppressors in regulating tumor-associated inflammationL Yang, M KarinTrends in Biochemical Sciences|October 1, 1992
Control of transcription factors by signal transduction pathways: the beginning of the endM Karin, T SmealThe Journal of Biological Chemistry|May 18, 1999
Stimulation of Elk1 transcriptional activity by mitogen-activated protein kinases is negatively regulated by protein phosphatase 2B (calcineurin)J Tian, M KarinGenes & Development|March 1, 1993
JunB differs from c-Jun in its DNA-binding and dimerization domains, and represses c-Jun by formation of inactive heterodimersT Deng, M KarinCell|July 17, 1987
A pituitary-specific trans-acting factor can stimulate transcription from the growth hormone promoter in extracts of nonexpressing cellsM Bodner, M KarinMatrix (Stuttgart, Germany). Supplement|January 1, 1992
Specific members of the Jun protein family regulate collagenase expression in response to various extracellular stimuliP Angel, M KarinPageof 350