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Alcohol (Fayetteville, N.Y.)|March 1, 1993
Omega-conotoxin increases sleep time following ethanol injectionL M Brown, J S Sims, P Randall, et al.Alcohol (Fayetteville, N.Y.)|May 1, 1990
Ethanol and neuronal calcium channelsS W Leslie, L M Brown, J E Dildy, et al.Brain Research|May 3, 1991
The effects of chronic ethanol exposure on N-methyl-D-aspartate-stimulated overflow of [3H]catecholamines from rat brainL M Brown, S W Leslie, R A GonzalesAlcohol (Fayetteville, N.Y.)|November 1, 1992
Alcohol inhibition of NMDA-stimulated catecholamine efflux in aging brainL M Brown, R D Trent, T W Jones, et al.Neurobiology of Aging|May 1, 1991
N-methyl-D-aspartate mediated responses decrease with age in Fischer 344 rat brainR A Gonzales, L M Brown, T W Jones, et al.Ohio Medicine : Journal of the Ohio State Medical Association|April 1, 1989
Outpatient metabolic evaluation of patients with recurrent kidney stonesS W LeslieRecent Developments in Alcoholism : an Official Publication of the American Medical Society on Alcoholism, the Research Society on Alcoholism, and the National Council on Alcoholism|January 1, 1987
Calcium channels. Interactions with ethanol and other sedative-hypnotic drugsS W LeslieBehavioural Brain Research|May 1, 1987
Monosodium L-glutamate lesions reduce susceptibility to hypoglycemic feeding and convulsionsJ F Lorden, J S SimsBehavioural Brain Research|June 1, 1988
Ventromedial hypothalamic and paraventricular nucleus lesions damage a common system to produce hyperphagiaJ E Cox, J S SimsBehavioural Brain Research|December 1, 1986
Effect of paraventricular nucleus lesions on body weight, food intake and insulin levelsJ S Sims, J F LordenPageof 104