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The Journal of Experimental Medicine|March 1, 1994
Terminal complement proteins C5b-9 release basic fibroblast growth factor and platelet-derived growth factor from endothelial cellsL R Benzaquen, A Nicholson-Weller, J A HalperinImmunologic Research|January 1, 1993
Membrane signaling by complement C5b-9, the membrane attack complexA Nicholson-Weller, J A HalperinComplement and Inflammation|January 1, 1989
Paroxysmal nocturnal hemoglobinuria. A complement-mediated diseaseJ A Halperin, A Nicholson-WellerThe Journal of Clinical Investigation|May 1, 1993
Terminal complement complex C5b-9 stimulates mitogenesis in 3T3 cellsJ A Halperin, A Taratuska, A Nicholson-WellerThe Journal of Clinical Investigation|May 1, 1989
Ca2+-activated K+ efflux limits complement-mediated lysis of human erythrocytesJ A Halperin, C Brugnara, A Nicholson-WellerBlood|January 1, 1993
Transient changes in erythrocyte membrane permeability are induced by sublytic amounts of the complement membrane attack complex (C5b-9)J A Halperin, A Taratuska, M Rynkiewicz, et al.The Journal of Clinical Investigation|August 1, 1988
Complement induces a transient increase in membrane permeability in unlysed erythrocytesJ A Halperin, A Nicholson-Weller, C Brugnara, et al.Nature Medicine|June 1, 1995
Clotrimazole inhibits cell proliferation in vitro and in vivoL R Benzaquen, C Brugnara, H R Byers, et al.Molecular Medicine (Cambridge, Mass.)|November 1, 1996
The transient pore formed by homologous terminal complement complexes functions as a bidirectional route for the transport of autocrine and paracrine signals across human cell membranesJ A Acosta, L R Benzaquen, D J Goldstein, et al.Current Opinion in Immunology|February 27, 1999
C1q-binding proteins and C1q receptorsA Nicholson-Weller, L B KlicksteinPageof 8