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Pflugers Archiv : European Journal of Physiology|November 29, 2007
H2O 2-stimulated Ca2+ influx via TRPM2 is not the sole determinant of subsequent cell deathJenny A Wilkinson, Jason L Scragg, John P Boyle, et al.
The Journal of Biological Chemistry|July 4, 2008
Carbon monoxide inhibits L-type Ca2+ channels via redox modulation of key cysteine residues by mitochondrial reactive oxygen speciesJason L Scragg, Mark L Dallas, Jenny A Wilkinson, et al.
Antioxidants & Redox Signaling|November 15, 2011
Modulation of ion channels by hydrogen sulfideChris Peers, Claudia C Bauer, John P Boyle, et al.
Biochemical and Biophysical Research Communications|May 4, 2004
Testosterone is a potent inhibitor of L-type Ca(2+) channelsJason L Scragg, Richard D Jones, Kevin S Channer, et al.
Advances in Experimental Medicine and Biology|August 26, 2015
Heme Oxygenase-1 Influences Apoptosis via CO-mediated Inhibition of K+ ChannelsMoza M Al-Owais, Mark L Dallas, John P Boyle, et al.
The Journal of Biological Chemistry|April 13, 2005
Hypoxic augmentation of Ca2+ channel currents requires a functional electron transport chainStephen T Brown, Jason L Scragg, John P Boyle, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|October 21, 2004
Alzheimer's amyloid peptides mediate hypoxic up-regulation of L-type Ca2+ channelsJason L Scragg, Ian M Fearon, John P Boyle, et al.
Annals of the New York Academy of Sciences|October 23, 2009
Hypoxia and neurodegenerationChris Peers, Mark L Dallas, Hannah E Boycott, et al.
Cell Death & Disease|June 16, 2017
Heme oxygenase-1 derived carbon monoxide suppresses Aβ<sub>1-42</sub> toxicity in astrocytesNishani T Hettiarachchi, John P Boyle, Mark L Dallas, et al.
The Journal of Biological Chemistry|April 11, 2014
Inhibition of the cardiac Na⁺ channel Nav1.5 by carbon monoxideJacobo Elies, Mark L Dallas, John P Boyle, et al.
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