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Lance R Thomas

Showing results (1-10 of 23) with videos related to

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Advances in Cancer Research|June 28, 2011
Proteolytic control of the oncoprotein transcription factor MycLance R Thomas, William P Tansey
The Journal of Biological Chemistry|July 11, 2002
Regulation of Fas-associated death domain interactions by the death effector domain identified by a modified reverse two-hybrid screenLance R Thomas, David J Stillman, Andrew Thorburn
Advances in Experimental Medicine and Biology|September 8, 2009
Dynamic regulation of antigen receptor gene assemblyLance R Thomas, Robin Milley Cobb, Eugene M Oltz
Biotechniques|March 3, 2004
pRS yeast vectors with a LYS2 markerPeter Eriksson, Lance R Thomas, Andrew Thorburn, et al.
The Journal of Biological Chemistry|September 29, 2004
The C-terminal tails of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) and Fas receptors have opposing functions in Fas-associated death domain (FADD) recruitment and can regulate agonist-specific mechanisms of receptor activationLance R Thomas, Ronald L Johnson, John C Reed, et al.
Nature Immunology|October 24, 2006
Control of thymocyte development and recombination-activating gene expression by the zinc finger protein Zfp608Feng Zhang, Lance R Thomas, Eugene M Oltz, et al.
Cancer Research|September 19, 2015
The MYC-WDR5 Nexus and CancerLance R Thomas, Audra M Foshage, April M Weissmiller, et al.
The Journal of Biological Chemistry|June 3, 2004
Direct binding of Fas-associated death domain (FADD) to the tumor necrosis factor-related apoptosis-inducing ligand receptor DR5 is regulated by the death effector domain of FADDLance R Thomas, Adrianna Henson, John C Reed, et al.
The Journal of Biological Chemistry|August 2, 2007
Tumor-derived mutations in the TRAIL receptor DR5 inhibit TRAIL signaling through the DR4 receptor by competing for ligand bindingLianghua Bin, Jacqueline Thorburn, Lance R Thomas, et al.
Molecular & Cellular Oncology|March 12, 2020
Targeting MYC through WDR5Lance R Thomas, Clare M Adams, Stephen W Fesik, et al.
Pageof 3

Showing results (1-10 of 23) with videos related to

Sort By:
Pageof 3
Advances in Cancer Research|June 28, 2011
Proteolytic control of the oncoprotein transcription factor MycLance R Thomas, William P Tansey
The Journal of Biological Chemistry|July 11, 2002
Regulation of Fas-associated death domain interactions by the death effector domain identified by a modified reverse two-hybrid screenLance R Thomas, David J Stillman, Andrew Thorburn
Advances in Experimental Medicine and Biology|September 8, 2009
Dynamic regulation of antigen receptor gene assemblyLance R Thomas, Robin Milley Cobb, Eugene M Oltz
Biotechniques|March 3, 2004
pRS yeast vectors with a LYS2 markerPeter Eriksson, Lance R Thomas, Andrew Thorburn, et al.
The Journal of Biological Chemistry|September 29, 2004
The C-terminal tails of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) and Fas receptors have opposing functions in Fas-associated death domain (FADD) recruitment and can regulate agonist-specific mechanisms of receptor activationLance R Thomas, Ronald L Johnson, John C Reed, et al.
Nature Immunology|October 24, 2006
Control of thymocyte development and recombination-activating gene expression by the zinc finger protein Zfp608Feng Zhang, Lance R Thomas, Eugene M Oltz, et al.
Cancer Research|September 19, 2015
The MYC-WDR5 Nexus and CancerLance R Thomas, Audra M Foshage, April M Weissmiller, et al.
The Journal of Biological Chemistry|June 3, 2004
Direct binding of Fas-associated death domain (FADD) to the tumor necrosis factor-related apoptosis-inducing ligand receptor DR5 is regulated by the death effector domain of FADDLance R Thomas, Adrianna Henson, John C Reed, et al.
The Journal of Biological Chemistry|August 2, 2007
Tumor-derived mutations in the TRAIL receptor DR5 inhibit TRAIL signaling through the DR4 receptor by competing for ligand bindingLianghua Bin, Jacqueline Thorburn, Lance R Thomas, et al.
Molecular & Cellular Oncology|March 12, 2020
Targeting MYC through WDR5Lance R Thomas, Clare M Adams, Stephen W Fesik, et al.
Pageof 3