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Lawrence Banks

Showing results (61-70 of 173) with videos related to

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Oncogene|July 27, 2005
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2ADavid Pim, Paola Massimi, Stephen M Dilworth, et al.
The Biochemical Journal|September 30, 2011
PDZ domains: the building blocks regulating tumorigenesisVanitha Krishna Subbaiah, Christian Kranjec, Miranda Thomas, et al.
Virology|December 26, 2014
High risk HPV E6 oncoproteins impair the subcellular distribution of the four and a half LIM-only protein 2 (FHL2)Joaquin Manzo-Merino, Paola Massimi, Lawrence Banks, et al.
Tumour Virus Research|March 15, 2021
Human papillomavirus E6 and E7: What remains?Arushi Vats, Oscar Trejo-Cerro, Miranda Thomas, et al.
Virology Journal|January 21, 2011
Analysis of the PDZ binding specificities of Influenza A virus NS1 proteinsMiranda Thomas, Christian Kranjec, Kazunori Nagasaka, et al.
Plos Pathogens|May 23, 2019
The HPV-18 E7 CKII phospho acceptor site is required for maintaining the transformed phenotype of cervical tumour-derived cellsOm Basukala, Suruchi Mittal, Paola Massimi, et al.
Journal of Cellular Biochemistry|April 23, 2002
Human papillomavirus-16 E7 protein inhibits the DNA interaction of the TATA binding transcription factorEdio Maldonado, María Eugenia Cabrejos, Lawrence Banks, et al.
Virology Journal|June 4, 2008
Comparison of p53 and the PDZ domain containing protein MAGI-3 regulation by the E6 protein from high-risk human papillomavirusesJulia Ainsworth, Miranda Thomas, Lawrence Banks, et al.
Tumour Virus Research|March 14, 2024
HPV16 E7 modulates the cell surface expression of MET and CD109 via the AP2 complexOscar Trejo-Cerro, Om Basukala, Michael P Myers, et al.
Scientific Reports|January 18, 2018
HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entryJustyna Broniarczyk, Nadja Ring, Paola Massimi, et al.
Pageof 18

Showing results (61-70 of 173) with videos related to

Sort By:
Pageof 18
Oncogene|July 27, 2005
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2ADavid Pim, Paola Massimi, Stephen M Dilworth, et al.
The Biochemical Journal|September 30, 2011
PDZ domains: the building blocks regulating tumorigenesisVanitha Krishna Subbaiah, Christian Kranjec, Miranda Thomas, et al.
Virology|December 26, 2014
High risk HPV E6 oncoproteins impair the subcellular distribution of the four and a half LIM-only protein 2 (FHL2)Joaquin Manzo-Merino, Paola Massimi, Lawrence Banks, et al.
Tumour Virus Research|March 15, 2021
Human papillomavirus E6 and E7: What remains?Arushi Vats, Oscar Trejo-Cerro, Miranda Thomas, et al.
Virology Journal|January 21, 2011
Analysis of the PDZ binding specificities of Influenza A virus NS1 proteinsMiranda Thomas, Christian Kranjec, Kazunori Nagasaka, et al.
Plos Pathogens|May 23, 2019
The HPV-18 E7 CKII phospho acceptor site is required for maintaining the transformed phenotype of cervical tumour-derived cellsOm Basukala, Suruchi Mittal, Paola Massimi, et al.
Journal of Cellular Biochemistry|April 23, 2002
Human papillomavirus-16 E7 protein inhibits the DNA interaction of the TATA binding transcription factorEdio Maldonado, María Eugenia Cabrejos, Lawrence Banks, et al.
Virology Journal|June 4, 2008
Comparison of p53 and the PDZ domain containing protein MAGI-3 regulation by the E6 protein from high-risk human papillomavirusesJulia Ainsworth, Miranda Thomas, Lawrence Banks, et al.
Tumour Virus Research|March 14, 2024
HPV16 E7 modulates the cell surface expression of MET and CD109 via the AP2 complexOscar Trejo-Cerro, Om Basukala, Michael P Myers, et al.
Scientific Reports|January 18, 2018
HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entryJustyna Broniarczyk, Nadja Ring, Paola Massimi, et al.
Pageof 18