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M A McDevitt

Showing results (11-20 of 24) with videos related to

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The EMBO Journal|November 1, 1986
Sequences capable of restoring poly(A) site function define two distinct downstream elementsM A McDevitt, R P Hart, W W Wong, et al.
Mutation Research|February 6, 2007
Fluorescent in situ hybridization (FISH) in bone marrow and peripheral blood of leukemia patients: implications for occupational surveillanceM A McDevitt, M Condon, J Stamberg, et al.
Genes & Development|May 1, 1988
Multiple factors are required for poly(A) addition to a mRNA 3' endM A McDevitt, G M Gilmartin, W H Reeves, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 24, 1997
A "knockdown" mutation created by cis-element gene targeting reveals the dependence of erythroid cell maturation on the level of transcription factor GATA-1M A McDevitt, R A Shivdasani, Y Fujiwara, et al.
Genes & Development|July 1, 1987
Relative position and strengths of poly(A) sites as well as transcription termination are critical to membrane versus secreted mu-chain expression during B-cell developmentG Galli, J W Guise, M A McDevitt, et al.
Archives of Biochemistry and Biophysics|April 1, 1984
Malic enzyme and fatty acid synthase in the uropygial gland and liver of embryonic and neonatal ducklings. Tissue-specific regulation of gene expressionA G Goodridge, R A Jenik, M A McDevitt, et al.
Development (Cambridge, England)|May 20, 1999
Different sequence requirements for expression in erythroid and megakaryocytic cells within a regulatory element upstream of the GATA-1 geneP Vyas, M A McDevitt, A B Cantor, et al.
The Journal of Biological Chemistry|March 25, 1982
Molecular cloning of gene sequences for avian fatty acid synthase and evidence for nutritional regulation of fatty acid synthase mRNA concentrationS M Morris, J H Nilson, R A Jenik, et al.
Leukemia|September 18, 2009
TP53 mutations in myeloid malignancies are either homozygous or hemizygous due to copy number-neutral loss of heterozygosity or deletion of 17pM Jasek, L P Gondek, N Bejanyan, et al.
Leukemia|June 7, 2013
Granulocyte colony-stimulating factor receptor T595I (T618I) mutation confers ligand independence and enhanced signalingH M Mehta, T Glaubach, A Long, et al.
Pageof 3

Showing results (11-20 of 24) with videos related to

Sort By:
Pageof 3
The EMBO Journal|November 1, 1986
Sequences capable of restoring poly(A) site function define two distinct downstream elementsM A McDevitt, R P Hart, W W Wong, et al.
Mutation Research|February 6, 2007
Fluorescent in situ hybridization (FISH) in bone marrow and peripheral blood of leukemia patients: implications for occupational surveillanceM A McDevitt, M Condon, J Stamberg, et al.
Genes & Development|May 1, 1988
Multiple factors are required for poly(A) addition to a mRNA 3' endM A McDevitt, G M Gilmartin, W H Reeves, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 24, 1997
A "knockdown" mutation created by cis-element gene targeting reveals the dependence of erythroid cell maturation on the level of transcription factor GATA-1M A McDevitt, R A Shivdasani, Y Fujiwara, et al.
Genes & Development|July 1, 1987
Relative position and strengths of poly(A) sites as well as transcription termination are critical to membrane versus secreted mu-chain expression during B-cell developmentG Galli, J W Guise, M A McDevitt, et al.
Archives of Biochemistry and Biophysics|April 1, 1984
Malic enzyme and fatty acid synthase in the uropygial gland and liver of embryonic and neonatal ducklings. Tissue-specific regulation of gene expressionA G Goodridge, R A Jenik, M A McDevitt, et al.
Development (Cambridge, England)|May 20, 1999
Different sequence requirements for expression in erythroid and megakaryocytic cells within a regulatory element upstream of the GATA-1 geneP Vyas, M A McDevitt, A B Cantor, et al.
The Journal of Biological Chemistry|March 25, 1982
Molecular cloning of gene sequences for avian fatty acid synthase and evidence for nutritional regulation of fatty acid synthase mRNA concentrationS M Morris, J H Nilson, R A Jenik, et al.
Leukemia|September 18, 2009
TP53 mutations in myeloid malignancies are either homozygous or hemizygous due to copy number-neutral loss of heterozygosity or deletion of 17pM Jasek, L P Gondek, N Bejanyan, et al.
Leukemia|June 7, 2013
Granulocyte colony-stimulating factor receptor T595I (T618I) mutation confers ligand independence and enhanced signalingH M Mehta, T Glaubach, A Long, et al.
Pageof 3