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Gene Therapy|June 27, 2008
Automated analysis of viral integration sites in gene therapy research using the SeqMap web resourceB Peters, S Dirscherl, J Dantzer, et al.The Journal of Infectious Diseases|December 8, 1998
Are reactive oxygen species involved in the pathogenesis of murine cerebral malaria?L A Sanni, S Fu, R T Dean, et al.Proceedings of the National Academy of Sciences of the United States of America|July 8, 1999
O2 sensing is preserved in mice lacking the gp91 phox subunit of NADPH oxidaseS L Archer, H L Reeve, E Michelakis, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 5, 2000
Inhibition of the Rac1 GTPase protects against nonlethal ischemia/reperfusion-induced necrosis and apoptosis in vivoM Ozaki, S S Deshpande, P Angkeow, et al.Stroke|November 22, 1997
Ischemic stroke injury is reduced in mice lacking a functional NADPH oxidaseC E Walder, S P Green, W C Darbonne, et al.Science (New York, N.Y.)|March 4, 2000
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidaseA Vazquez-Torres, Y Xu, J Jones-Carson, et al.Blood|September 15, 1992
Splice site mutations are a common cause of X-linked chronic granulomatous diseaseM de Boer, B G Bolscher, M C Dinauer, et al.Proceedings of the National Academy of Sciences of the United States of America|December 15, 1991
Point mutation in the cytoplasmic domain of the neutrophil p22-phox cytochrome b subunit is associated with a nonfunctional NADPH oxidase and chronic granulomatous diseaseM C Dinauer, E A Pierce, R W Erickson, et al.Human Gene Therapy|August 7, 1998
Correction of respiratory burst activity in X-linked chronic granulomatous cells to therapeutically relevant levels after gene transfer into bone marrow CD34+ cellsS Becker, S Wasser, M Hauses, et al.The Journal of Experimental Medicine|December 1, 1993
Restitution of superoxide generation in autosomal cytochrome-negative chronic granulomatous disease (A22(0) CGD)-derived B lymphocyte cell lines by transfection with p22phax cDNAF E Maly, C C Schuerer-Maly, L Quilliam, et al.Pageof 8