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The British Journal of Ophthalmology|August 25, 2006
Socioeconomic status, systolic blood pressure and intraocular pressure: the Tanjong Pagar StudyJ L Y Yip, T Aung, T-Y Wong, et al.
The British Journal of Ophthalmology|December 4, 2012
Spectral domain optical coherence tomography imaging of the aqueous outflow structures in normal participants of the EPIC-Norfolk Eye StudyAlexander C Day, David F Garway-Heath, David C Broadway, et al.
Ophthalmology. Glaucoma|May 14, 2022
Acute Angle-Closure Attacks Are Uncommon in Primary Angle-Closure Suspects after Pharmacologic Mydriasis: The Zhongshan Angle-Closure Prevention TrialDavid S Friedman, Dolly S Chang, Yuzhen Jiang, et al.
BMJ Open Ophthalmology|August 24, 2024
Estimating biological age from retinal imaging: a scoping reviewMichaela Joan Grimbly, Sheri-Michelle Koopowitz, Ruiye Chen, et al.
Clinical Endocrinology|November 20, 2013
MRI-based abnormalities in young adults at risk of adverse bone health due to childhood-onset metabolic & endocrine conditionsC McComb, A Harpur, C Yacoubian, et al.
Molecular Microbiology|December 20, 2005
Fibronectin-binding proteins of Staphylococcus aureus mediate activation of human platelets via fibrinogen and fibronectin bridges to integrin GPIIb/IIIa and IgG binding to the FcgammaRIIa receptorJ Ross Fitzgerald, Anthony Loughman, Fiona Keane, et al.
Ophthalmology|November 29, 2011
Risk of acute angle closure and changes in intraocular pressure after pupillary dilation in Asian subjects with narrow anglesRaghavan Lavanya, Mani Baskaran, Rajesh S Kumar, et al.
Archives of Ophthalmology (Chicago, Ill. : 1960)|April 13, 2005
Anterior chamber depth and the risk of primary angle closure in 2 East Asian populationsTin Aung, Winifred P Nolan, David Machin, et al.
The FEBS Journal|September 27, 2006
Staphylococcus aureus protein A binding to von Willebrand factor A1 domain is mediated by conserved IgG binding regionsMaghnus O'Seaghdha, Carina J van Schooten, Steven W Kerrigan, et al.
Scandinavian Journal of Immunology|February 13, 2001
Sirolimus interferes with the innate response to bacterial products in human whole blood by attenuation of IL-10 productionP F Jørgensen, J E Wang, M Almlöf, et al.
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