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M Karch

Showing results (71-80 of 196) with videos related to

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Molecular Psychiatry|September 21, 2023
Long non-coding RNA SNHG8 drives stress granule formation in tauopathiesReshma Bhagat, Miguel A Minaya, Arun Renganathan, et al.
Nature Protocols|May 17, 2023
Generation of iPSC-derived human forebrain organoids assembling bilateral eye primordiaElke Gabriel, Walid Albanna, Giovanni Pasquini, et al.
Medrxiv : the Preprint Server for Health Sciences|February 20, 2025
A novel lncRNA FAM151B-DT regulates autophagy and degradation of aggregation prone proteinsArun Renganathan, Miguel A Minaya, Matthew Broder, et al.
Translational Psychiatry|December 15, 2018
Integrative system biology analyses of CRISPR-edited iPSC-derived neurons and human brains reveal deficiencies of presynaptic signaling in FTLD and PSPShan Jiang, Natalie Wen, Zeran Li, et al.
Plos One|February 25, 2011
Fine mapping of genetic variants in BIN1, CLU, CR1 and PICALM for association with cerebrospinal fluid biomarkers for Alzheimer's diseaseJohn S K Kauwe, Carlos Cruchaga, Celeste M Karch, et al.
Stem Cell Reports|October 3, 2017
Scalable Production of iPSC-Derived Human Neurons to Identify Tau-Lowering Compounds by High-Content ScreeningChao Wang, Michael E Ward, Robert Chen, et al.
Human Molecular Genetics|June 6, 2014
Coding variants in TREM2 increase risk for Alzheimer's diseaseSheng Chih Jin, Bruno A Benitez, Celeste M Karch, et al.
Nature Communications|March 31, 2026
A pathogenic Tau mutation drives autophagy-lysosome dysfunction that limits Tau degradation in a model of frontotemporal dementiaFarzaneh S Mirfakhar, Jacob A Marsh, Chihiro Sato, et al.
Biorxiv : the Preprint Server for Biology|December 25, 2025
Integrative Genomic and Functional Analyses Reveal NINL as a Modulator of Tau AggregationSamantha K Swift, Guangming Huang, J Nicholas Cochran, et al.
Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|January 28, 2025
Evaluating pathogenicity of variants of unknown significance in APP, PSEN1, and PSEN2Jacob A Marsh, Guangming Huang, Kevin Bowling, et al.
Pageof 20

Showing results (71-80 of 196) with videos related to

Sort By:
Pageof 20
Molecular Psychiatry|September 21, 2023
Long non-coding RNA SNHG8 drives stress granule formation in tauopathiesReshma Bhagat, Miguel A Minaya, Arun Renganathan, et al.
Nature Protocols|May 17, 2023
Generation of iPSC-derived human forebrain organoids assembling bilateral eye primordiaElke Gabriel, Walid Albanna, Giovanni Pasquini, et al.
Medrxiv : the Preprint Server for Health Sciences|February 20, 2025
A novel lncRNA FAM151B-DT regulates autophagy and degradation of aggregation prone proteinsArun Renganathan, Miguel A Minaya, Matthew Broder, et al.
Translational Psychiatry|December 15, 2018
Integrative system biology analyses of CRISPR-edited iPSC-derived neurons and human brains reveal deficiencies of presynaptic signaling in FTLD and PSPShan Jiang, Natalie Wen, Zeran Li, et al.
Plos One|February 25, 2011
Fine mapping of genetic variants in BIN1, CLU, CR1 and PICALM for association with cerebrospinal fluid biomarkers for Alzheimer's diseaseJohn S K Kauwe, Carlos Cruchaga, Celeste M Karch, et al.
Stem Cell Reports|October 3, 2017
Scalable Production of iPSC-Derived Human Neurons to Identify Tau-Lowering Compounds by High-Content ScreeningChao Wang, Michael E Ward, Robert Chen, et al.
Human Molecular Genetics|June 6, 2014
Coding variants in TREM2 increase risk for Alzheimer's diseaseSheng Chih Jin, Bruno A Benitez, Celeste M Karch, et al.
Nature Communications|March 31, 2026
A pathogenic Tau mutation drives autophagy-lysosome dysfunction that limits Tau degradation in a model of frontotemporal dementiaFarzaneh S Mirfakhar, Jacob A Marsh, Chihiro Sato, et al.
Biorxiv : the Preprint Server for Biology|December 25, 2025
Integrative Genomic and Functional Analyses Reveal NINL as a Modulator of Tau AggregationSamantha K Swift, Guangming Huang, J Nicholas Cochran, et al.
Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|January 28, 2025
Evaluating pathogenicity of variants of unknown significance in APP, PSEN1, and PSEN2Jacob A Marsh, Guangming Huang, Kevin Bowling, et al.
Pageof 20