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Blood|September 18, 1997
The hemoglobin-deficit mouse: analysis of phenotype and hematopoiesis in the transplant modelM L Bloom, K L Simon-StoosExperimental Hematology|January 29, 1999
The hemoglobin-deficit mouse: cure of the anemia following bone marrow transplantation with normal marrowK L Simon-Stoos, M L BloomClinical Immunology (Orlando, Fla.)|September 25, 1999
Expression in transgenic mice of dominant interfering Fas mutations: a model for human autoimmune lymphoproliferative syndromeY Choi, V R Ramnath, A S Eaton, et al.Orthopedics|January 1, 1985
Slipped capital femoral epiphysis. An assessment of treatment modalitiesM L Bloom, A H CrawfordBlood|November 1, 1993
Complete nucleotide sequence of the murine erythroid beta-spectrin cDNA and tissue-specific expression in normal and jaundiced miceM L Bloom, C S Birkenmeier, J E BarkerClinical and Experimental Immunology|July 1, 1976
Heterophile antibodies in pathologic human sera resembling antibodies stimulated by foreign species seraR Kasukawa, K Kano, M L Bloom, et al.Cancer Research|May 1, 1983
Use of phenobarbital and high doses of 1-(2-chloroethyl)-3-cyclohexyl-1-nitrosourea in the treatment of brain tumor-bearing miceP J Muller, C H Tator, M L BloomProceedings of the National Academy of Sciences of the United States of America|October 11, 1994
The murine mutation jaundiced is caused by replacement of an arginine with a stop codon in the mRNA encoding the ninth repeat of beta-spectrinM L Bloom, T M Kaysser, C S Birkenmeier, et al.Human Gene Therapy|June 13, 1998
Murine bone marrow expressing the neomycin resistance gene has no competitive disadvantage assessed in vivoT Wu, M L Bloom, J M Yu, et al.Pageof 3