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Free Radical Biology & Medicine|November 26, 2002
Is oxidative damage the fundamental pathogenic mechanism of Alzheimer's and other neurodegenerative diseases?George Perry, Akihiko Nunomura, Keisuke Hirai, et al.
Biochemistry|August 25, 2004
Differential inhibition of six copper amine oxidases by a family of 4-(aryloxy)-2-butynamines: evidence for a new mode of inactivationKimberly M O'Connell, David B Langley, Eric M Shepard, et al.
The Journal of Biological Chemistry|September 18, 2009
Catabolism of 4-hydroxyacids and 4-hydroxynonenal via 4-hydroxy-4-phosphoacyl-CoAsGuo-Fang Zhang, Rajan S Kombu, Takhar Kasumov, et al.
Health Physics|July 30, 2011
FDA-sunlamp recommended Maximum Timer Interval And Exposure Schedule: consensus ISO/CIE dose equivalenceJohn C Dowdy, Eugene A Czako, Michael E Stepp, et al.
Neurochemistry International|February 19, 2002
Amyloid-beta, tau alterations and mitochondrial dysfunction in Alzheimer disease: the chickens or the eggs?Mark A Smith, Kelly L Drew, Akihiko Nunomura, et al.
Free Radical Research|April 10, 2013
Neurofilaments are the major neuronal target of hydroxynonenal-mediated protein cross-linksE A Perry, R J Castellani, P I Moreira, et al.
Biochemistry|January 14, 2004
Copper mediates dityrosine cross-linking of Alzheimer's amyloid-betaCraig S Atwood, George Perry, Hong Zeng, et al.
The Journal of Biological Chemistry|December 26, 2001
High molecular weight neurofilament proteins are physiological substrates of adduction by the lipid peroxidation product hydroxynonenalTakafumi Wataya, Akihiko Nunomura, Mark A Smith, et al.
Free Radical Biology & Medicine|December 6, 2011
Hydroxynonenal-generated crosslinking fluorophore accumulation in Alzheimer disease reveals a dichotomy of protein turnoverXiongwei Zhu, Rudy J Castellani, Paula I Moreira, et al.
Metabolism: Clinical and Experimental|May 27, 2008
25-Hydroxyvitamin D, cholesterol, and ultraviolet irradiationLaura D Carbone, E William Rosenberg, Elizabeth A Tolley, et al.
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