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Science (New York, N.Y.)|May 3, 1996
Molecular genetic insights into cardiovascular diseaseM T Keating, M C SanguinettiCurrent Opinion in Genetics & Development|June 1, 1996
Pathophysiology of ion channel mutationsM T Keating, M C SanguinettiThe American Journal of Physiology|March 1, 1997
Single HERG delayed rectifier K+ channels expressed in Xenopus oocytesA Zou, M E Curran, M T Keating, et al.Cell|April 21, 1995
A mechanistic link between an inherited and an acquired cardiac arrhythmia: HERG encodes the IKr potassium channelM C Sanguinetti, C Jiang, M E Curran, et al.The Journal of Biological Chemistry|April 3, 1999
Long QT syndrome-associated mutations in the Per-Arnt-Sim (PAS) domain of HERG potassium channels accelerate channel deactivationJ Chen, A Zou, I Splawski, et al.Proceedings of the National Academy of Sciences of the United States of America|March 5, 1996
Spectrum of HERG K+-channel dysfunction in an inherited cardiac arrhythmiaM C Sanguinetti, M E Curran, P S Spector, et al.Circulation Research|March 1, 1996
Class III antiarrhythmic drugs block HERG, a human cardiac delayed rectifier K+ channel. Open-channel block by methanesulfonanilidesP S Spector, M E Curran, M T Keating, et al.Circulation|August 29, 2001
Long-QT syndrome-associated missense mutations in the pore helix of the HERG potassium channelF D Huang, J Chen, M Lin, et al.Nature Genetics|November 14, 1997
Mutations in the hminK gene cause long QT syndrome and suppress IKs functionI Splawski, M Tristani-Firouzi, M H Lehmann, et al.The Journal of General Physiology|May 1, 1996
Fast inactivation causes rectification of the IKr channelP S Spector, M E Curran, A Zou, et al.Pageof 13