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Drug Discovery Today|September 10, 2023
Metabolic reprogramming: Unveiling the therapeutic potential of targeted therapies against kidney diseaseShubhangi Saxena, Neha Dagar, Vishwadeep Shelke, et al.
Drug Discovery Today|December 12, 2022
Promising novel therapeutic targets for kidney disease: Emphasis on kidney-specific proteinsTathambika Tejeswini Sen, Ajinath Kale, Maciej Lech, et al.
International Immunology|June 30, 2010
Quantitative expression of RIG-like helicase, NOD-like receptor and inflammasome-related mRNAs in humans and miceMaciej Lech, Alejandro Avila-Ferrufino, Veronika Skuginna, et al.
International Journal of Molecular Sciences|September 6, 2012
Quantitative expression of C-type lectin receptors in humans and miceMaciej Lech, Heni Eka Susanti, Christoph Römmele, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 27, 2022
MCP-Induced Protein 1 Participates in Macrophage-Dependent Endotoxin ToleranceMarta Wadowska, Ewelina Dobosz, Anna Golda, et al.
Journal of Innate Immunity|August 12, 2016
MCPIP-1, Alias Regnase-1, Controls Epithelial Inflammation by Posttranscriptional Regulation of IL-8 ProductionEwelina Dobosz, Mateusz Wilamowski, Maciej Lech, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 6, 2010
Ischemia reperfusion induces IFN regulatory factor 4 in renal dendritic cells, which suppresses postischemic inflammation and prevents acute renal failureSaraswati Lassen, Maciej Lech, Christoph Römmele, et al.
Journal of the American Society of Nephrology : JASN|April 27, 2007
Inhibition of Toll-like receptor-7 (TLR-7) or TLR-7 plus TLR-9 attenuates glomerulonephritis and lung injury in experimental lupusRahul D Pawar, Allam Ramanjaneyulu, Onkar P Kulkarni, et al.
Journal of the American Society of Nephrology : JASN|November 4, 2006
Ligands to nucleic acid-specific toll-like receptors and the onset of lupus nephritisRahul D Pawar, Prashant S Patole, Andreas Ellwart, et al.
Frontiers in Cellular and Infection Microbiology|May 13, 2017
Inactive Gingipains from P. gingivalis Selectively Skews T Cells toward a Th17 Phenotype in an IL-6 Dependent MannerIzabela Glowczyk, Alicia Wong, Barbara Potempa, et al.
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