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Cell Cycle (Georgetown, Tex.)|May 7, 2015
Apoptotic effects of high-dose rapamycin occur in S-phase of the cell cycleMahesh Saqcena, Deven Patel, Deepak Menon, et al.
Plos One|August 27, 2013
Amino acids and mTOR mediate distinct metabolic checkpoints in mammalian G1 cell cycleMahesh Saqcena, Deepak Menon, Deven Patel, et al.
The Journal of Biological Chemistry|January 31, 2015
Reciprocal regulation of AMP-activated protein kinase and phospholipase DSuman Mukhopadhyay, Mahesh Saqcena, Amrita Chatterjee, et al.
The Journal of Biological Chemistry|December 14, 2016
A Late G1 Lipid Checkpoint That Is Dysregulated in Clear Cell Renal Carcinoma CellsDeven Patel, Darin Salloum, Mahesh Saqcena, et al.
The Journal of Biological Chemistry|May 31, 2011
Phospholipase D mediates nutrient input to mammalian target of rapamycin complex 1 (mTORC1)Limei Xu, Darin Salloum, Phil S Medlin, et al.
The Journal of Clinical Endocrinology and Metabolism|December 13, 2021
Characterization of Subtypes of BRAF-Mutant Papillary Thyroid Cancer Defined by Their Thyroid Differentiation ScoreLaura Boucai, Venkatraman Seshan, Michelle Williams, et al.
Cell Cycle (Georgetown, Tex.)|November 11, 2011
High-dose rapamycin induces apoptosis in human cancer cells by dissociating mTOR complex 1 and suppressing phosphorylation of 4E-BP1Paige Yellen, Mahesh Saqcena, Darin Salloum, et al.
Research Square|July 28, 2023
Cooperative Genomic Lesions in HRAS-Mutant Cancers Predict Resistance to Farnesyltransferase InhibitorsAradhya Nigam, Gnana Krishnamoorthy, Walid Chatila, et al.
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