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Methods in Molecular Biology (Clifton, N.J.)|August 26, 2020
In Vivo Binding of Recombination Proteins to Non-DSB DNA Lesions and to Replication ForksRomán González-Prieto, María J Cabello-Lobato, Félix PradoThe EMBO Journal|April 9, 2013
Rad51 replication fork recruitment is required for DNA damage toleranceRomán González-Prieto, Ana M Muñoz-Cabello, María J Cabello-Lobato, et al.Nucleic Acids Research|October 11, 2014
Defective histone supply causes condensin-dependent chromatin alterations, SAC activation and chromosome decatenation impairmentMarina Murillo-Pineda, María J Cabello-Lobato, Marta Clemente-Ruiz, et al.Epigenetics & Chromatin|July 24, 2019
Crosstalk between chromatin structure, cohesin activity and transcriptionDouglas Maya-Miles, Eloísa Andújar, Mónica Pérez-Alegre, et al.Cell Reports|July 28, 2021
Physical interactions between MCM and Rad51 facilitate replication fork lesion bypass and ssDNA gap filling by non-recombinogenic functionsMaría J Cabello-Lobato, Cristina González-Garrido, María I Cano-Linares, et al.Pageof 1