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Proceedings of the National Academy of Sciences of the United States of America|May 18, 2012
Critical role of soluble amyloid-β for early hippocampal hyperactivity in a mouse model of Alzheimer's diseaseMarc Aurel Busche, Xiaowei Chen, Horst A Henning, et al.Nature Neuroscience|November 10, 2015
Decreased amyloid-β and increased neuronal hyperactivity by immunotherapy in Alzheimer's modelsMarc Aurel Busche, Christine Grienberger, Aylin D Keskin, et al.Science (New York, N.Y.)|September 20, 2008
Clusters of hyperactive neurons near amyloid plaques in a mouse model of Alzheimer's diseaseMarc Aurel Busche, Gerhard Eichhoff, Helmuth Adelsberger, et al.Neuron|April 18, 2026
Interpreting human sleep activity through neural contrastive learningZhongtao Chen, Hui Zheng, Jianyang Zhou, et al.Nature Neuroscience|December 19, 2018
Tau impairs neural circuits, dominating amyloid-β effects, in Alzheimer models in vivoMarc Aurel Busche, Susanne Wegmann, Simon Dujardin, et al.Nature Communications|July 10, 2024
β-amyloid monomer scavenging by an anticalin protein prevents neuronal hyperactivity in mouse models of Alzheimer's DiseaseBenedikt Zott, Lea Nästle, Christine Grienberger, et al.Alzheimer'S Research & Therapy|July 9, 2021
Aη-α and Aη-β peptides impair LTP ex vivo within the low nanomolar range and impact neuronal activity in vivoMaria Mensch, Jade Dunot, Sandy M Yishan, et al.Nature Neuroscience|June 4, 2026
Aberrant tau accumulation caused by MAPT mutations induces early pathological changes in axonal transport that are rescued by p38α inhibitionEdoardo Moretto, Anna Masato, Chiara Panzi, et al.Proceedings of the National Academy of Sciences of the United States of America|July 26, 2017
BACE inhibition-dependent repair of Alzheimer's pathophysiologyAylin D Keskin, Maja Kekuš, Helmuth Adelsberger, et al.JPRAS Open|January 19, 2026
Neurolymphatic clearance in neurodegenerative disease: Emerging mechanisms and potential translational strategiesAdriano Fabi, André S Alves, Albert Neutzner, et al.Pageof 3