Showing results (1-10 of 17) with videos related to

Sort By:
Pageof 2
Biomedicines|March 28, 2025
Exploring Disparities in Atherosclerosis Comorbidity with Aortic AneurysmAksana N Kucher, Iuliia A Koroleva, Maria S Nazarenko
Biochemistry. Biokhimiia|March 11, 2024
Pathogenetic Significance of Long Non-Coding RNAs in the Development of Thoracic and Abdominal Aortic AneurysmsAksana N Kucher, Iuliia A Koroleva, Maria S Nazarenko
International Journal of Molecular Sciences|August 26, 2022
Meier-Gorlin Syndrome: Clinical Misdiagnosis, Genetic Testing and Functional Analysis of <i>ORC6</i> Mutations and the Development of a Prenatal TestMaria S Nazarenko, Iuliia V Viakhireva, Mikhail Y Skoblov, et al.
Data in Brief|December 17, 2021
Human exome sequence data in support of somatic mosaicism in carotid atherosclerosisAlexei A Sleptcov, Alexei A Zarubin, Polina M Bogaychuk, et al.
International Journal of Molecular Sciences|June 10, 2023
Apoptosis Genes as a Key to Identification of Inverse Comorbidity of Huntington's Disease and CancerElena Yu Bragina, Densema E Gomboeva, Olga V Saik, et al.
Frontiers in Bioscience (Scholar Edition)|March 27, 2024
A New Leu714Arg Variant in the Converter Domain of <i>MYH7</i> is Associated with a Severe Form of Familial Hypertrophic CardiomyopathyMaria V Golubenko, Elena N Pavlyukova, Ramil R Salakhov, et al.
International Journal of Molecular Sciences|December 23, 2022
Application of Long-Read Nanopore Sequencing to the Search for Mutations in Hypertrophic CardiomyopathyRamil R Salakhov, Maria V Golubenko, Nail R Valiakhmetov, et al.
Scientific Reports|January 26, 2017
Genomic structural variations for cardiovascular and metabolic comorbidityMaria S Nazarenko, Aleksei A Sleptcov, Igor N Lebedev, et al.
Plos One|April 10, 2015
A comparison of genome-wide DNA methylation patterns between different vascular tissues from patients with coronary heart diseaseMaria S Nazarenko, Anton V Markov, Igor N Lebedev, et al.
Stem Cell Research|January 9, 2022
Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLRIrina S Zakharova, Alexander I Shevchenko, Narek A Tmoyan, et al.
Pageof 2