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Diabetes|October 29, 2002
Reduction of the accumulation of advanced glycation end products by ACE inhibition in experimental diabetic nephropathyJosephine M Forbes, Mark E Cooper, Vicki Thallas, et al.
BMJ Open Diabetes Research & Care|December 3, 2014
Retinopathy and clinical outcomes in patients with type 2 diabetes mellitus, chronic kidney disease, and anemiaNatalie A Bello, Marc A Pfeffer, Hicham Skali, et al.
Journal of Cardiovascular Translational Research|June 20, 2012
Oxidative stress, Nox isoforms and complications of diabetes--potential targets for novel therapiesMona Sedeek, Augusto C Montezano, Richard L Hebert, et al.
American Journal of Physiology. Renal Physiology|December 18, 2009
Disparate effects on renal and oxidative parameters following RAGE deletion, AGE accumulation inhibition, or dietary AGE control in experimental diabetic nephropathyAdeline L Y Tan, Karly C Sourris, Brooke E Harcourt, et al.
Clinical Science (London, England : 1979)|December 11, 2012
Association of dietary sodium intake with atherogenesis in experimental diabetes and with cardiovascular disease in patients with Type 1 diabetesChris Tikellis, Raelene J Pickering, Despina Tsorotes, et al.
American Journal of Nephrology|August 31, 2011
Advanced glycation urinary protein-bound biomarkers and severity of diabetic nephropathy in manMelinda T Coughlan, Sheila K Patel, George Jerums, et al.
Antioxidants & Redox Signaling|September 24, 2013
NADPH oxidase, NOX1, mediates vascular injury in ischemic retinopathyJennifer L Wilkinson-Berka, Devy Deliyanti, Indrajeetsinh Rana, et al.
Journal of Hypertension|February 27, 2010
Advanced glycation end-products induce vascular dysfunction via resistance to nitric oxide and suppression of endothelial nitric oxide synthaseAino Soro-Paavonen, Wei-Zeng Zhang, Kylie Venardos, et al.
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