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Molecular & Cellular Oncology|June 17, 2016
Inhibitors of cell cycle checkpoints and DNA replication cause different responses in normal versus malignant urothelial cellsMark MeuthMolecular Biology of the Cell|November 11, 2005
Chk1 and p21 cooperate to prevent apoptosis during DNA replication fork stressRene Rodriguez, Mark MeuthCarcinogenesis|February 14, 2003
Reduced apoptotic response to camptothecin in CHO cells deficient in XRCC3John M Hinz, Thomas Helleday, Mark MeuthMolecular Biology of the Cell|January 8, 2010
Enhanced H2AX phosphorylation, DNA replication fork arrest, and cell death in the absence of Chk1Mary E Gagou, Pedro Zuazua-Villar, Mark MeuthHuman Molecular Genetics|April 2, 2003
A naturally occurring mutation in an ATP-binding domain of the recombination repair gene XRCC3 ablates its function without causing cancer susceptibilitySaeed Rafii, Annika Lindblom, Malcolm Reed, et al.International Journal of Oncology|September 7, 2011
Treatment with the Chk1 inhibitor Gö6976 enhances cisplatin cytotoxicity in SCLC cellsRuth Thompson, Mark Meuth, Penella Woll, et al.Human Molecular Genetics|October 2, 2004
ATM is required for the cellular response to thymidine induced replication fork stressEmma Bolderson, Jennifer Scorah, Thomas Helleday, et al.Stem Cell Research & Therapy|January 27, 2016
Apoptosis and failure of checkpoint kinase 1 activation in human induced pluripotent stem cells under replication stressJoelle A Desmarais, Christian Unger, Ivan Damjanov, et al.Plos Genetics|January 3, 2009
ATR and Chk1 suppress a caspase-3-dependent apoptotic response following DNA replication stressKatie Myers, Mary E Gagou, Pedro Zuazua-Villar, et al.Pageof 4