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Cancers|August 7, 2021
Primary Aldosteronism: Metabolic Reprogramming and the Pathogenesis of Aldosterone-Producing AdenomasSiyuan Gong, Martina Tetti, Martin Reincke, et al.
Molecular and Cellular Endocrinology|May 26, 2016
The long N-terminus of the human monocarboxylate transporter 8 is a target of ubiquitin-dependent proteasomal degradation which regulates protein expression and oligomerization capacityDenise Zwanziger, Mathias Schmidt, Jana Fischer, et al.
Plos One|November 11, 2011
Differential modulation of Beta-adrenergic receptor signaling by trace amine-associated receptor 1 agonistsGunnar Kleinau, Juliane Pratzka, Daniela Nürnberg, et al.
Experimental and Clinical Endocrinology & Diabetes : Official Journal, German Society of Endocrinology [And] German Diabetes Association|September 11, 2018
Inherited Forms of Primary Hyperaldosteronism: New Genes, New Phenotypes and Proposition of A New ClassificationLuis Gustavo Perez-Rivas, Tracy Ann Williams, Martin Reincke
European Journal of Endocrinology|May 1, 2007
Screening for mutations in transcription factors in a Czech cohort of 170 patients with congenital and early-onset hypothyroidism: identification of a novel PAX8 mutation in dominantly inherited early-onset non-autoimmune hypothyroidismEva Al Taji, Heike Biebermann, Zdenka Límanová, et al.
The Journal of Clinical Endocrinology and Metabolism|March 6, 2021
Approach to the Patient Treated with Steroidogenesis InhibitorsFrederic Castinetti, Lynnette K Nieman, Martin Reincke, et al.
Vitamins and Hormones|February 4, 2026
Adrenal venous sampling interpretation and molecular imaging as localization tool in primary aldosteronismSinan Deniz, Matthias Oettle, Markus Kroiss, et al.
Endocrine Practice : Official Journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists|August 8, 2018
PATHOGENESIS OF CUSHING DISEASE: AN UPDATE ON THE GENETICS OF CORTICOTROPINOMASAdriana Albani, Luis G Perez-Rivas, Martin Reincke, et al.
The Journal of Clinical Endocrinology and Metabolism|November 20, 2013
Identification of PENDRIN (SLC26A4) mutations in patients with congenital hypothyroidism and "apparent" thyroid dysgenesisPeter Kühnen, Serap Turan, Sebastian Fröhler, et al.
The Journal of Biological Chemistry|May 1, 2008
G13-dependent activation of MAPK by thyrotropinThomas R H Büch, Heike Biebermann, Hermann Kalwa, et al.
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