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The EMBO Journal|December 18, 2002
PDK1-dependent activation of atypical PKC leads to degradation of the p21 tumour modifier proteinMary T Scott, Angela Ingram, Kathryn L BallFrontiers in Cell and Developmental Biology|August 6, 2019
Epigenetic Reprogramming and Emerging Epigenetic Therapies in CMLJane Bugler, Ross Kinstrie, Mary T Scott, et al.The Journal of Cell Biology|August 4, 2010
Akt/PKB suppresses DNA damage processing and checkpoint activation in late G2Naihan Xu, Nadia Hegarat, Elizabeth J Black, et al.Developmental Cell|February 6, 2007
Chk1 is required for spindle checkpoint functionGeorge Zachos, Elizabeth J Black, Mark Walker, et al.Nature Protocols|December 19, 2014
Antibody-based detection of protein phosphorylation status to track the efficacy of novel therapies using nanogram protein quantities from stem cells and cell linesMark Aspinall-O'Dea, Andrew Pierce, Francesca Pellicano, et al.Molecular and Cellular Biology|March 7, 2007
The MDM2 ubiquitination signal in the DNA-binding domain of p53 forms a docking site for calcium calmodulin kinase superfamily membersAshley L Craig, Jennifer A Chrystal, Jennifer A Fraser, et al.Stem Cells (Dayton, Ohio)|May 9, 2014
The antiproliferative activity of kinase inhibitors in chronic myeloid leukemia cells is mediated by FOXO transcription factorsFrancesca Pellicano, Mary T Scott, G Vignir Helgason, et al.Science Translational Medicine|June 12, 2024
Nutrient-sensitizing drug repurposing screen identifies lomerizine as a mitochondrial metabolism inhibitor of chronic myeloid leukemiaAhmed Khalaf, Lucie de Beauchamp, Eric Kalkman, et al.Nature Communications|January 21, 2024
Activating p53 abolishes self-renewal of quiescent leukaemic stem cells in residual CML diseaseMary T Scott, Wei Liu, Rebecca Mitchell, et al.Cancer Discovery|September 16, 2016
Epigenetic Reprogramming Sensitizes CML Stem Cells to Combined EZH2 and Tyrosine Kinase InhibitionMary T Scott, Koorosh Korfi, Peter Saffrey, et al.Pageof 2