Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Marzia Fumagalli

Showing results (1-10 of 14) with videos related to

Pageof 2
Sort By:
Nature Cell Biology|August 4, 2009
SASPense and DDRama in cancer and ageingMarzia Fumagalli, Fabrizio d'Adda di Fagagna
Trends in Cell Biology|November 6, 2007
Breaking news: high-speed race ends in arrest--how oncogenes induce senescenceRaffaella Di Micco, Marzia Fumagalli, Fabrizio d'Adda di Fagagna
Plos One|October 24, 2014
Stable cellular senescence is associated with persistent DDR activationMarzia Fumagalli, Francesca Rossiello, Chiara Mondello, et al.
Ecancermedicalscience|May 28, 2015
The SUMO conjugating enzyme UBC9 as a biomarker for cervical HPV infectionsDomenico Mattoscio, Chiara Casadio, Marzia Fumagalli, et al.
Current Opinion in Genetics & Development|August 9, 2014
Irreparable telomeric DNA damage and persistent DDR signalling as a shared causative mechanism of cellular senescence and ageingFrancesca Rossiello, Utz Herbig, Maria Pia Longhese, et al.
European Journal of Radiology|November 29, 2024
The translation of in-house imaging AI research into a medical device ensuring ethical and regulatory integrityFilippo Pesapane, Mathias K Hauglid, Marzia Fumagalli, et al.
Cell Cycle (Georgetown, Tex.)|November 13, 2008
DNA damage response activation in mouse embryonic fibroblasts undergoing replicative senescence and following spontaneous immortalizationRaffaella Di Micco, Angelo Cicalese, Marzia Fumagalli, et al.
Journal of Cellular Physiology|November 7, 2003
Inhibition of proliferation and induction of apoptosis in breast cancer cells by the epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor ZD1839 ('Iressa') is independent of EGFR expression levelManuela Campiglio, Alberta Locatelli, Clelia Olgiati, et al.
Frontiers in Molecular Biosciences|September 12, 2022
Standard operating procedures for biobank in oncologyGiuseppina Bonizzi, Lorenzo Zattoni, Maria Capra, et al.
Nature|December 1, 2006
Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replicationRaffaella Di Micco, Marzia Fumagalli, Angelo Cicalese, et al.
Pageof 2

Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
Nature Cell Biology|August 4, 2009
SASPense and DDRama in cancer and ageingMarzia Fumagalli, Fabrizio d'Adda di Fagagna
Trends in Cell Biology|November 6, 2007
Breaking news: high-speed race ends in arrest--how oncogenes induce senescenceRaffaella Di Micco, Marzia Fumagalli, Fabrizio d'Adda di Fagagna
Plos One|October 24, 2014
Stable cellular senescence is associated with persistent DDR activationMarzia Fumagalli, Francesca Rossiello, Chiara Mondello, et al.
Ecancermedicalscience|May 28, 2015
The SUMO conjugating enzyme UBC9 as a biomarker for cervical HPV infectionsDomenico Mattoscio, Chiara Casadio, Marzia Fumagalli, et al.
Current Opinion in Genetics & Development|August 9, 2014
Irreparable telomeric DNA damage and persistent DDR signalling as a shared causative mechanism of cellular senescence and ageingFrancesca Rossiello, Utz Herbig, Maria Pia Longhese, et al.
European Journal of Radiology|November 29, 2024
The translation of in-house imaging AI research into a medical device ensuring ethical and regulatory integrityFilippo Pesapane, Mathias K Hauglid, Marzia Fumagalli, et al.
Cell Cycle (Georgetown, Tex.)|November 13, 2008
DNA damage response activation in mouse embryonic fibroblasts undergoing replicative senescence and following spontaneous immortalizationRaffaella Di Micco, Angelo Cicalese, Marzia Fumagalli, et al.
Journal of Cellular Physiology|November 7, 2003
Inhibition of proliferation and induction of apoptosis in breast cancer cells by the epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor ZD1839 ('Iressa') is independent of EGFR expression levelManuela Campiglio, Alberta Locatelli, Clelia Olgiati, et al.
Frontiers in Molecular Biosciences|September 12, 2022
Standard operating procedures for biobank in oncologyGiuseppina Bonizzi, Lorenzo Zattoni, Maria Capra, et al.
Nature|December 1, 2006
Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replicationRaffaella Di Micco, Marzia Fumagalli, Angelo Cicalese, et al.
Pageof 2