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Therapeutische Umschau. Revue Therapeutique|October 31, 2013
[Pediatric emergencies -- the essential, briefly, for general practitioners]Mascha K Rochat, Mario Gehri
American Journal of Medical Genetics. Part A|January 19, 2007
Long-term follow-up of a 26-year-old male with duplication of 16p: clinical report and reviewMascha K Rochat, Mariluce Riegel, Albert A Schinzel
World Journal of Gastroenterology|May 19, 2010
Disagreement between symptom-reflux association analysis parameters in pediatric gastroesophageal reflux disease investigationSamuel C Lüthold, Mascha K Rochat, Peter Bähler
Pediatric Pulmonology|August 21, 2015
Lack of concordance in parapneumonic effusion management in children in central EuropeGaudenz M Hafen, Andrea-Claudia Grenzbach, Alexander Moeller, et al.
The Journal of Biological Chemistry|May 17, 2002
Chenodeoxycholic acid and deoxycholic acid inhibit 11 beta-hydroxysteroid dehydrogenase type 2 and cause cortisol-induced transcriptional activation of the mineralocorticoid receptorAnita T Stauffer, Mascha K Rochat, Bernhard Dick, et al.
Acta Paediatrica (Oslo, Norway : 1992)|August 18, 2009
Association between peer relationship problems and childhood overweight/obesityAnja Boneberger, Rüdiger von Kries, Astrid Milde-Busch, et al.
The Journal of Allergy and Clinical Immunology|November 6, 2010
Allergic rhinitis as a predictor for wheezing onset in school-aged childrenMascha K Rochat, Sabina Illi, Markus J Ege, et al.
Plos One|January 16, 2013
Spirometry reference equations for central European populations from school age to old ageMascha K Rochat, Ruediger P Laubender, Daniela Kuster, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 11, 2003
Hypoxia causes down-regulation of 11 beta-hydroxysteroid dehydrogenase type 2 by induction of Egr-1Christian D Heiniger, Radina M Kostadinova, Mascha K Rochat, et al.
American Journal of Respiratory and Critical Care Medicine|January 12, 2013
A Th17- and Th2-skewed cytokine profile in cystic fibrosis lungs represents a potential risk factor for Pseudomonas aeruginosa infectionKerstin Tiringer, Angela Treis, Petra Fucik, et al.
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