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Neurological Sciences : Official Journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology|March 7, 2012
Proteomic identification of aldolase A as an autoantibody target in patients with atypical movement disordersDaniela Privitera, Valeria Corti, Massimo Alessio, et al.
Journal of Proteome Research|January 9, 2008
Serological immunoreactivity against colon cancer proteome varies upon disease progressionLucia De Monte, Francesca Sanvito, Stefano Olivieri, et al.
Journal of Proteomics|December 3, 2013
A possible role of transglutaminase 2 in the nucleus of INS-1E and of cells of human pancreatic isletsSara Sileno, Valentina D'Oria, Riccardo Stucchi, et al.
Molecular Neurodegeneration|November 6, 2015
Ceruloplasmin functional changes in Parkinson's disease-cerebrospinal fluidMarco Barbariga, Flavio Curnis, Annapaola Andolfo, et al.
Acta Diabetologica|March 3, 2012
Transglutaminase 2 transamidation activity during first-phase insulin secretion: natural substrates in INS-1ELucia Russo, Claudia Marsella, Giovanni Nardo, et al.
Cancer Research|March 17, 2007
Molecular cloning of hMena (ENAH) and its splice variant hMena+11a: epidermal growth factor increases their expression and stimulates hMena+11a phosphorylation in breast cancer cell linesFrancesca Di Modugno, Lucia DeMonte, Michele Balsamo, et al.
Oncotarget|August 13, 2016
Serological immune response against ADAM10 pro-domain is associated with favourable prognosis in stage III colorectal cancer patientsSheila María Álvarez-Fernández, Marco Barbariga, Luca Cannizzaro, et al.
Journal of Proteomics|March 19, 2013
Serological Proteome Analysis (SERPA) as a tool for the identification of new candidate autoantigens in type 1 diabetesOrnella Massa, Massimo Alessio, Lucia Russo, et al.
Plos One|January 7, 2011
The cooperation between hMena overexpression and HER2 signalling in breast cancerFrancesca Di Modugno, Marcella Mottolese, Lucia DeMonte, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 16, 2011
Ceruloplasmin oxidation, a feature of Parkinson's disease CSF, inhibits ferroxidase activity and promotes cellular iron retentionStefano Olivieri, Antonio Conti, Sandro Iannaccone, et al.
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