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Journal of Molecular and Cellular Cardiology|July 10, 2010
Normal passive viscoelasticity but abnormal myofibrillar force generation in human hypertrophic cardiomyopathyAnita C Hoskins, Adam Jacques, Sonya C Bardswell, et al.The Journal of Biological Chemistry|September 2, 2020
Receptor-independent modulation of cAMP-dependent protein kinase and protein phosphatase signaling in cardiac myocytes by oxidizing agentsSimon Diering, Konstantina Stathopoulou, Mara Goetz, et al.JCI Insight|October 18, 2019
Prelamin A mediates myocardial inflammation in dilated and HIV-associated cardiomyopathiesDaniel Brayson, Andrea Frustaci, Romina Verardo, et al.The FEBS Journal|February 17, 2022
CMYA5 is a novel interaction partner of FHL2 in cardiac myocytesKonstantina Stathopoulou, Josef Schnittger, Janice Raabe, et al.Development (Cambridge, England)|December 19, 2008
Prox1 maintains muscle structure and growth in the developing heartCatherine A Risebro, Richelle G Searles, Athalie A D Melville, et al.Journal of Molecular and Cellular Cardiology|June 19, 2026
Structural, biophysical and cellular assessment of filamin C M82K: A test case for VUS interpretation in cardiomyopathyMaya Noureddine, Bethany A I Jones, Oseloka C M Oliobi, et al.Nature Communications|June 30, 2016
MLP and CARP are linked to chronic PKCα signalling in dilated cardiomyopathyStephan Lange, Katja Gehmlich, Alexander S Lun, et al.Journal of Molecular and Cellular Cardiology|July 27, 2018
Mutant Muscle LIM Protein C58G causes cardiomyopathy through protein depletionMehroz Ehsan, Matthew Kelly, Charlotte Hooper, et al.Elife|September 24, 2021
Myopalladin knockout mice develop cardiac dilation and show a maladaptive response to mechanical pressure overloadMaria Carmela Filomena, Daniel L Yamamoto, Pierluigi Carullo, et al.BMC Medical Genomics|January 31, 2009
Muscle Research and Gene Ontology: New standards for improved data integrationErika Feltrin, Stefano Campanaro, Alexander D Diehl, et al.Pageof 20